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cyclin

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cy·clin

(sī'klin),
A class of proteins involved in regulation of the cell cycle.
Farlex Partner Medical Dictionary © Farlex 2012

cyclin

(sī′klĭn)
n.
Any of a class of proteins that fluctuate in concentration at specific points during the cell cycle and that regulate the cycle by binding to a kinase.
The American Heritage® Medical Dictionary Copyright © 2007, 2004 by Houghton Mifflin Company. Published by Houghton Mifflin Company. All rights reserved.

PCNA

A gene on chromosome 20pter-p12 that encodes a homotrimeric nuclear protein that is a cofactor of DNA polymerase delta. PCNA is ubiquinated in response to DNA damage and is involved in RAD6-dependent DNA repair.
Segen's Medical Dictionary. © 2012 Farlex, Inc. All rights reserved.
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References in periodicals archive
Almost all cases show BCL2 positivity and express cyclin D1 which is involved in G1 to S phase cell cycle progression through activation of cyclin-dependent kinases.
Kaldis, "Mammalian cell-cycle regulation: several cdks, numerous cyclins and diverse compensatory mechanisms," Oncogene, vol.
Marzluff, "Phosphorylation of threonine 61 by cyclin A/Cdk1 triggers degradation of stem-loop binding protein at the end of S phase," Molecular and Cellular Biology, vol.
In prior work, they showed that cyclin D1 regulates the non coding genome, and that the non-coding genome, in turn, regulates expression of cyclin D1.
One of the major trials would be the validation of the results obtained in the inducible expression approach of AKT using the conditional deletion system of Tsc2 gene, specifically in relation to the post-transcriptional modulation of the levels of cyclin D2 and D3 as well as the activity of cdk4.
Semiquantitative evaluation of labelings of cyclins (B1, D1) and E-cadherin in untreated and vanadyl sulphate treated MCF-7 cells.
The specific primers for cyclin D1 (forward: 5'-TAGCAGCGAGCAGCAGAGTC-3', reverse: 5'-TTTCCACTTCGCAGCACAGG-3', 174bp) and for Bcl-xL (forward: 5'-AGCTT TGAACAGGATACTTTTGTGG-3', reverse: 5'-GGTGGGAGGGTAGAGTGGAT-3', 183 bp) were designed according to the BatchPrimer3 software (NCBI Reference Sequence: NM_053056.2 and NM_001191.2, respectively) and synthesized by Sangon Biotech (Sangon, China).
Further correlation analysis indicated that Bax and caspase-3 gene expression were positively correlated with p21 mRNA level ([r.sub.Bax-spleen] = +0.861, [P.sub.Bax-spleen] = 0.006 ; [r.sub.caspase-3-spleen] = +0.874, [P.sub.caspase-3-spleen] = 0.005 ; [r.sub.caspase-3-thymus] = +0.776, [P.sub.caspase-3-thymus] = 0.024 ), but negatively associated with cyclin A2 mRNA expression ([r.sub.Bax-spleen] = -0.960, [P.sub.Bax-spleen] = 0.000**; [r.sub.Bax-thymus] = -0.905, [P.sub.Bax-thymus] = 0 002**; [r.sub.caspase-3-spleen] = -0.961, [P.sub.caspase-3-spleen] = 0.000** [r.sub.caspase-3-thymus] = -0.941, [P.sub.caspase-3-thymus] = 0.000**), which is consistent with the above-mentioned results that spermine administration enhanced cell proliferation.
However, in the present study we were interested to see if the differences in serum estradiol we previously observed had any impact on muscle estradiol, ER-[alpha] mRNA and protein expression, ER-DNA binding, and the subsequent effect on the expression of the ER target genes/markers of myogenic activity, Myo-D and cyclin D1.
Khandan Keyomarsi, professor of experimental radiation oncology, led a team that demonstrated how CDK4/6 and autophagy inhibitors synergistically induce cell senescence in Rb-positive cytoplasmic cyclin E-negative cancers.
It also arrested cycling cells at S and [G.sub.2]/M phases and affected cyclin B1, Bcl-2, and Ki67 expression.
Immunohistochemical stains were positive for vimentin (Figure 5), cyclin D1 (Figure 6), CD99 (Figure 7), TLE1 (Figure 8), and focally positive for Bcl- 2 (Figure 9) in the tumor cells.
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