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fluent aphasia

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aphasia

 [ah-fa´zhah]
a type of speech disorder consisting of a defect or loss of the power of expression by speech, writing, or signs, or of comprehension of spoken or written language, due to disease or injury of the brain centers, such as after stroke syndrome on the left side.
Patient Care. Aphasia is a complex phenomenon manifested in numerous ways. The recovery period is often very long, even months or years. Because communication is such a vital part of everyday living, loss of the ability to communicate with words, whether in speaking or writing, can profoundly affect the personality and behavior of a patient. Although aphasic persons usually require extensive treatment by specially trained speech patholigists or therapists, all persons concerned with the care of the patient should practice techniques that will help minimize frustration and improve communication with such patients.
amnestic aphasia anomic aphasia.
anomic aphasia inability to name objects, qualities, or conditions. Called also amnestic or nominal aphasia.
ataxic aphasia expressive aphasia.
auditory aphasia loss of ability to comprehend spoken language. Called also word deafness.
Broca's aphasia motor aphasia.
conduction aphasia aphasia due to a lesion of the pathway between the sensory and motor speech centers.
expressive aphasia motor aphasia.
fluent aphasia that in which speech is well articulated (usually 200 or more words per minute) and grammatically correct but is lacking in content and meaning.
global aphasia total aphasia involving all the functions that go to make up speech and communication.
jargon aphasia that with utterance of meaningless phrases, either neologisms or incoherently arranged known words.
mixed aphasia combined expressive and receptive aphasia.
motor aphasia aphasia in which there is impairment of the ability to speak and write, owing to a lesion in the insula and surrounding operculum including Broca's motor speech area. The patient understands written and spoken words but has difficulty uttering the words. See also receptive aphasia. Called also logaphasia and Broca's, expressive, or nonfluent aphasia.
nominal aphasia anomic aphasia.
nonfluent aphasia motor aphasia.
receptive aphasia inability to understand written, spoken, or tactile speech symbols, due to disease of the auditory and visual word centers, as in word blindness. See also motor aphasia. Called also logamnesia and sensory or Wernicke's aphasia.
sensory aphasia receptive aphasia.
visual aphasia alexia.
Wernicke's aphasia receptive aphasia.
Miller-Keane Encyclopedia and Dictionary of Medicine, Nursing, and Allied Health, Seventh Edition. © 2003 by Saunders, an imprint of Elsevier, Inc. All rights reserved.

sen·so·ry a·pha·si·a

aphasia with impairment in the comprehension of spoken and written words, associated with effortless, articulated, but paraphrastic speech and writing; malformed words, substitute words, and neologisms are characteristic. When condition severe and speech is incomprehensible, it is called jargon aphasia. The patient often appears unaware of the deficit.
Farlex Partner Medical Dictionary © Farlex 2012

re·cep·tive a·pha·si·a

(rĕ-sĕp'tiv ă-fā'zē-ă)
A condition including impairment in the comprehension of spoken and written words, associated with effortless, articulated, but paraphasic speech and writing; malformed words, substitute words, and neologisms are characteristic. When severe, and speech is incomprehensible, it is called jargon aphasia. The patient often appears unaware of this deficit. The lesion typically includes a portion of the superior temporal lobe.
Synonym(s): fluent aphasia, sensory aphasia, Wernicke aphasia.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012

fluent aphasia

A disorder of brain function in which articulation of speech and grammatical organization are preserved but comprehension and choice of words are defective. The condition is common after STROKE.
Collins Dictionary of Medicine © Robert M. Youngson 2004, 2005
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References in periodicals archive
In reviewing this book, I would like to discuss two critical issues which arise from the initial premise that fluent aphasia is a clearly definable entity to be separated from nonfluent aphasia.
My second issue concerns the analysis of grammatical impairments in fluent aphasia. Although Edwards provides us with an interesting and very detailed discussion of lexical and grammatical deficits in fluent aphasia, she like many other researchers is resistant to positing the same underlying deficit in grammatical processing by Broca's and Wernicke's aphasics, even if their patterns converge.
Though it may not have been Edwards' intention to provide a comprehensive portrayal of fluent aphasia and therefore my following criticism may not be entirely justified, I still find two chapters missing from her book which I feel are absolutely necessary in a monograph meant as a kind of textbook to provide students with a general introduction to fluent aphasia.
Participants with non-fluent aphasia had more difficulty with pause time in turn-taking and quantity/conciseness than the participants with fluent aphasia. Avent and Wertz (1996) reported no significant differences between adults with fluent aphasia and adults with non-fluent aphasia with regard to pragmatic performance, but did observe that adults with fluent aphasia performed slightly better than adults with non-fluent aphasia.
If, on the whole, individuals with non-fluent aphasia are considered to be better functional communicators and individuals with fluent aphasia are better or at least equal pragmatically, then an examination of the relationship between pragmatics and functional communication in relationship to language impairment is warranted.
Avent and Wertz (1996) reported that adults with fluent aphasia in their sample demonstrated a slightly higher level of pragmatic appropriateness than did adults with non-fluent aphasia.
Participant 1 was an 80-year-old right-handed male who was 5 years postonset of a left-hemisphere cerebrovascular accident (CVA) resulting in fluent aphasia. A computed tomography (CT) scan performed 3 years later revealed a left temporal-parietal lesion.
Participant 1 presented with a moderately severe fluent aphasia consistent with the pattern of Wernickes aphasia.
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