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pancuronium

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pancuronium

Anesthesiology A long-acting nondepolarizing muscle relaxing anesthetic Mechanism Competitive inhibition of acetylcholine at motor end plate Pros Metabolized by plasma cholinesterase or nonenzymatic pathways, thus better in multiorgan failure Cons Evokes histamine release, causing hypotension, tachycardia. See Muscle relaxant. Nondepolarizing agent. Cf Depolarizing agent.
McGraw-Hill Concise Dictionary of Modern Medicine. © 2002 by The McGraw-Hill Companies, Inc.
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References in periodicals archive
Lethal vaccinations such as potassium chloride, sodium thiopental and pancuronium bromide are injected in the patients suffering from various terminal illness such as neurological conditions, last stage of Alzheimer's, chronic lung problem and heart failures.
Among nondepolarizing agents, pancuronium, cisatracurium, and atracurium have all been studied in the ARDS population.
A study conducted by Dupuis et al (29) found vecuronium better than pancuronium for reduction of shivering because vecuronium was not shown to increase myocardial work and was associated with fewer complications.
Vecuronium bromide (Org NC 45) is a monoquaternary analogue of Pancuronium. This agent was synthesised by Savage in 1979 (Churchill Davidson HC), most potent of all the steroid muscle relaxants studied so far.
These are sodium thiopental, which depresses the central nervous system in 30 seconds, inducing a degree of unconsciousness that makes pain undetectable; pancuronium bromide, a relaxant which takes 30-45 seconds to induce paralysis and respiratory arrest; and potassium chloride, which stops the heart in around 30 seconds.
Vender, "Postanesthesia care unit recovery times and neuromuscular blocking drugs: a prospective study of orthopedic surgical patients randomized to receive pancuronium or rocuronium," Anesthesia and Analgesia, vol.
IV pancuronium was used, 6-hourly for 2 days, then as required when spasms occurred.
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