visceral obesity

a form of obesity due to excessive deposition of fat in the abdominal viscera and omentum, rather than subcutaneously, associated with dyslipidemia (for example, increased plasma triglyceride, low high-density lipoprotein cholesterol); perhaps due to accelerated lipolysis and mobilization of abdominal fatty acids by way of the portal vein; poses greater risk of diabetes mellitus, hypertension, metabolic syndrome, and cardiovascular disease than peripheral obesity does.

Synonym(s): abdominal obesity

vis·cer·al o·be·si·ty

(vis'ĕr-ăl ō-bē'si-tē)

Overweight due to excessive deposition of fat in the abdominal viscera and omentum, rather than subcutaneously, associated with dyslipidemia; poses greater risk of diabetes mellitus, hypertension, metabolic syndrome, and cardiovascular disease than peripheral obesity does.

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References in periodicals archive ?

Obesity-related adverse health consequences occur predominately in individuals with upper body fat accumulation, the detrimental distribution, commonly associated with visceral obesity.[3] Abdominal obesity is more common among South Asians than general adiposity in contrast to whites, who have only a slightly higher rate of abdominal adiposity, and blacks who actually have a lower rate of abdominal obesity than general obesity.[4] Abdominal obesity is seen with increased prevalence in South Asians even among those who have a body mass index (BMI) <25 kg/[m.sup.2].

Study of relationship between anthropometric parameters and heart rate-corrected QT interval (QTc) in normal body mass index Indian males with abdominal obesity

Maternal and Post-Weaning Exposure to a High Fat Diet Promotes Visceral Obesity and Hepatic Steatosis in Adult Rats.

Effects of maternal high fat intake during pregnancy and lactation on total cholesterol and adipose tissue in neonatal rats/Efeitos do consumo materno de dieta hiperlipidica durante a gestacao e lactacao no colesterol total e tecido adiposo de ratos neonatos

Imaging body composition in cancer patients: visceral obesity, sarcopenia and sarcopenic obesity may impact on clinical outcome.

Examining the association between adiposity and biochemical recurrence after radical prostatectomy

This means that, according to BMI, the obese patients were not necessarily those with visceral obesity. Figure 2 shows the patients with similar BMI but with visceral fat and subcutaneous fat.

Prediction of the Risk of Laparoscopy-Assisted Gastrectomy by Comparing Visceral Fat Area and Body Mass Index

Most mechanisms of sarcopenia are also associated with visceral obesity, which may lead to a vicious cycle of intricate interactions among risk factors.

Osteosarcopenic Visceral Obesity and Osteosarcopenic Subcutaneous Obesity, Two New Phenotypes of Sarcopenia: Prevalence, Metabolic Profile, and Risk Factors

Obesity, especially visceral obesity, could increase the risk of type 2 diabetes, hypertension, dyslipidemia, and atherosclerosis, suggesting that obese patients with type 2 diabetes are at high risk for cardiovascular diseases [1].

Exenatide with Metformin Ameliorated Visceral Adiposity and Insulin Resistance

The purpose of this study was to explore whether NAFLD, another accurate indicator for visceral obesity and insulin resistance, could be used as a surrogate for MetS diagnosis in the normal WC population.

Fatty Liver as a Potential Surrogate for Waist Circumference in the Diagnosis of Metabolic Syndrome: A Population-Based Study among Chinese Adults

Even at supposedly normal BMIs of 23 to 25, there can be hidden fatness observed, particularly in the abdomen (visceral obesity).

Why diabetics face the risk of having cardiovascular disease

The definition of visceral obesity was VFA exceeding 130 [cm.sup.2] [19].

Role of Nampt and Visceral Adiposity in Esophagogastric Junction Adenocarcinoma

Participants with FTO risk allele (A) had significantly higher odds of being overweight/obese (OR: 0.19 (0.10-0.36), p < 0.001), central obesity (OR: 0.33 (0.15-0.71), p = 0.005), and visceral obesity (OR: 0.12 (0.06-0.23), p < 0.001) than individuals with the T allele only (Table 3).

FTO Gene Associates and Interacts with Obesity Risk, Physical Activity, Energy Intake, and Time Spent Sitting: Pilot Study in a Nigerian Population

While certain amounts of WAT are required for survival and health, central or visceral obesity has been linked to the etiology of chronic diseases such as cardiovascular disease, type 2 diabetes, and cancer [2, 3].

A Single Bout of Fasting (24 h) Reduces Basal Cytokine Expression and Minimally Impacts the Sterile Inflammatory Response in the White Adipose Tissue of Normal Weight F344 Rats

Despres, "Pathophysiology of human visceral obesity: an update," Physiological Reviews, vol.

Serum Vaspin Concentration in Elderly Type 2 Diabetes Mellitus Patients with Differing Body Mass Index: A Cross-Sectional Study