ventricular preload

ven·tric·u·lar pre·load

formerly, the end-diastolic pressure stretching the ventricular walls, which determines the end-diastolic fiber length at the onset of ventricular contraction, or some other measure of this load on the muscle fibers before contraction; now, more rigorously expressed in terms of the wall stress at this moment, related to the tension per unit cross-sectional area in the ventricular muscle fibers (calculated by Laplace law from internal radius and pressure modified by wall thickness) that balances this transmural pressure at the moment before contraction begins.
Synonym(s): preload (2)

ven·tric·u·lar pre·load

(ven-trik'yū-lăr prē'lōd)
The pressure stretching the ventricular walls at the onset of ventricular contraction, expressed in terms of the wall stress at this moment, related to the tension per unit cross-sectional area in the ventricular muscle fibers that balances this transmural pressure at the moment before contraction begins.
Synonym(s): preload (2) .

ven·tric·u·lar pre·load

(ven-trik'yū-lăr prē'lōd)
The pressure stretching the ventricular walls at the onset of ventricular contraction.
Synonym(s): preload (2) .
References in periodicals archive ?
Correlation among right ventricular preload, afterload and ejection fraction in mitral valve disease: Radionuclide, echocardiographic and hemodynamic evaluation.
The potential haemodynamic derangement associated with right ventricular infarction renders the patient's unusually sensitive to diminished ventricular preload.
One study showed that LV diastolic function may influence the increase in left ventricular preload as a result of CKD progression.
The researchers concluded that "PVI may serve as a reliable estimate of cardiac preload status in patients undergoing OLT, explicitly, higher PVI values correlated with lower RVEDVI values," so that "an increase in ventricular preload status could be inferred from a decrease in PVI during OLT.
The pneumoperitoneum-induced increases in abdominal pressure may compress the inferior vena cava, resulting in decreased right ventricular preload.
Venous return to the heart directly influences ventricular preload.
As a result, left ventricular preload is decreased and cardiac output falls.
The decreased transmural pressure increases the intraluminal pressure, which "pushes" the blood from pulmonary vessels into the left ventricle and increases left ventricular preload.
However, these changes lead to a reduction in LV compliance affecting the early diastolic filling, making the left ventricular preload more dependent on atrial contraction.
Patients unresponsive to medical therapy could be considered for atrial septostomy, which creates a right to left shunt and thus decompresses the right ventricle and increases left ventricular preload.

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