vasoactive substance

vasoactive substance

Any of a group of circulating substances that regulate vascular tone, causing either vasodilation–ANP, kinins, and VIP, or vasoconstriction–angiotensin II, epinephrine, norepinephrine, vasopressin
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Therefore, two explanations may be suggested to explain our results: (a) the longer physical exercise duration was sufficient to increase the vasoactive substance concentration (e.g., which has high release threshold) and/or enable/disable nervous system that can influence Q and PVR, causing SBP and DBP to decrease; and (b) the longer physical exercise duration was able to elicit an increase in other vasoactive substances and/or enable/disable nervous system, thus leading to a simultaneous biological activity of blood pressure controls.
In case of unilateral placenta, the absence or insufficient trophoblastic invasion of arteries on contralateral side would make them retain their sensitivity to vasoactive substance during pregnancy.
Probably, the release of vasoactive substance such as CGRP and Substance P (SP) which upon activation of A[delta]- and C-fibres via the axon reflex lead to vasodilatation of small vessels, thus increasing the blood flow.
Endothelial injury leads to an increase of potent vasoactive substance such as endothelin, fibronectin and thromboxane.
These vessels are responsive to vasoactive substance but morphologically functionally and histologically they are peculiar.
For patients with sudden sensorineural hearing loss, clinicians should not order routine computed tomography of the head in the initial evaluation, should not obtain routine laboratory tests, and should not routinely prescribe antivirals, thrombolytics, vasodilators, or vasoactive substances.
This metastasis allows serotonin and other vasoactive substances to bypass hepatic metabolic degradation, resulting in the aforementioned symptoms.
First, RCVS occurring in the setting of blood pressure fluctuation, use of sympathomimetic vasoactive substances, and pheochromocytoma supports the role of sympathetic overactivity in its pathogenesis.
Various causative mechanism includes alteration in plasminogen modulators, increased level of matrix metalloproteinases, alteration in vasoactive substances and impact on angiogenesis.
[1] published a Cochrane review regarding vasodilators and vasoactive substances for ISSHL.
When sepsis occurs, a network is established between the endogenous inflammatory mediators including vasoactive substances, cytokines, chemokines, oxygen radicals, acute phase reactants, bioactive lipid, plasma enzyme system products, and fibrinolytic pathways.

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