uteroplacental insufficiency


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Related to uteroplacental insufficiency: IUGR

uteroplacental insufficiency

Inadequate blood flow through the placental intervillous spaces to enable sufficient transmission of nutrients, oxygen, and fetal wastes. It may be caused by diminished maternal cardiac output due to anemia, heart disease, regional anesthesia, or supine hypotension; vasoconstriction due to chronic or pregnancy-related hypertension or uterine overstimulation; vasospasm due to pregnancy-induced hypertension; vascular sclerosis due to maternal diabetes or collagen disease; or intrauterine infection. It increases the risk for intrauterine growth retardation.
See also: insufficiency
References in periodicals archive ?
Uteroplacental insufficiency increases apoptosis and alters p53 gene methylation in the full-term IUGR rat kidney.
Myocardial gene expression of glucose transporter 1 and glucose transporter 4 in response to uteroplacental insufficiency in the rat.
This can be attributed to asymetrical IUGR which accounts for 70-80% in which uteroplacental insufficiency (PIH) was the most common cause.
Repeat Doppler on second week on Day 15 of admission was suggestive of uteroplacental insufficiency (Increased RI of umbilical artery) and absent uterine artery flow.
Severe degree of uteroplacental insufficiency in group B than in group A could be the reason for greater number of IUGR babies in group B.