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found that fifty percent of patients with BOO had overactive bladder (OAB).1 OAB, a clinical syndrome caused by abnormal afferent signals and detrusor overactivity, used to be called unstable bladder, the main symptoms of which were urgent urination, uroclepsia, frequent micturition, increased urination at night, and it would seriously influence patients' life quality.2,3 Study showed that the more obvious prostatic median lobe hyperplasia was and more serious BOO symptoms were,4 the severer OAB symptoms were; the reason why OAB occurred was secondary changes of stability, elasticity and sensitivity of bladder which was caused by BOO.
Meta-analysis of uroclepsia, weight gain, upper respiratory infections, tremor, dry mouth, insomnia, and electroencephalography changes did not differ between the groups (Supplemental Figure 3).
It can become large and displace the urinary bladder or rectum, leading to various symptoms, including repeated urinary tract infection, epididymitis, hematuresis, pyuria, uroclepsia, oligospermia, uroschesis, and constipation.