The presence in the urine of urobilins in excessive amounts, formed mainly from hemoglobin.
Farlex Partner Medical Dictionary © Farlex 2012


, urobilinogenuria (yūr'ō-bil'i-nyū'rē-ă, -i-nō-jĕ-nyūrē-ă)
Excess urobilin in urine, formed mainly from hemoglobin.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012
References in periodicals archive ?
In malaria patients from Colombia, hemoglobinuria (86%), urobilinuria (66%), proteinuria (54%), hematuria (43%), and bilirubinuria (39%) were associated with dark urine; renal dysfunction was identified in these patients [7].
Present in 147 (24.3%) of 605 patients, bilirubinuria was associated with urobilinuria, proteinuria, and increased specific gravity (P < 0.05).
Urobilinuria was identified in 184 (30.6%) of 602 patients; it was associated with higher bilirubin in urine and proteinuria (P < 0.05).
This alteration was associated with bilirubinuria, urobilinuria, blood, and increased specific gravity (P < 0.05) and was related to higher serum BUN, TB, DB, ALT, and aspartatetransaminase (AST) values and lower platelet counts.
falciparum cases showed higher frequency of ketonuria, proteinuria, and urobilinuria (P < 0.005, Chi-square test).
The most common urinalysis alterations in the 620 malaria patients were increased specific gravity (41.2%), proteinuria (39.2%), urobilinuria (30.6%), blood (29.8%), bilirubinuria (24.3%), ketones (22.7%), and leukocytes (14.86%).
In our patients, proteinuria was associated with increased TB and DB serum levels, with increased risk of liver, kidney, and lung complications, and other alterations in urinalysis including bilirubinuria, urobilinuria, ketonuria, and glycosuria.
Associations were found among serum ALT and bilirubinuria (P = 0.01), urobilinuria (P = 0.006), and proteinuria (P = 0.001); this contributes to explaining a relationship between liver complication and bilirubinuria (OR 2.1, CI 1.3-3.3), urobilinuria (OR 2.7, CI 1.7-4.5), and proteinuria (OR 1.5, CI 1.0-2.3).
This technique can be used for distinguishing uroporphyrinuria because of the in vitro polymerization of porphobilinogen and congenital erythropoietic porphyria; only the latter is complicated by hemolytic anemia and urobilinuria. In addition to a direct identification of urobilins, our method also provides important corroborative evidence for fecal identification by demonstrating the coexistence of dicarboxylic porphyrins, which are the bacterial degradation products of protoporphyrin [9].