The poster presentation includes data demonstrating urate reduction in a urate oxidase knock-out mouse model, an animal model of severe hyperuricemia with kidney damage due to urate crystal
Monosodium urate crystal
formation is reversible, and crystals will dissolve when the sUA level drops below the limit of solubility (~6.8 mg/dL).
examined the frequency of urate crystal
deposition in 92 patients using dual-energy computer tomography .
Gout, referred to as the urate crystal
deposition disease, is a painful inflammatory arthritis that can drop the quality of life of affected individuals and restrict them to a wheelchair if not treated on time.
In patients with gout, sodium urate crystal
deposition can occur in subcutaneous tissues.
Colchicine administration has blocked CappD-crystal and urate crystal
Gout is a metabolic disease of monosodium urate crystal
deposition and the peak age incidence occurs at 30-50 years .
In this article, we report a chronic gout patient with serious CTS secondary to monosodium urate crystal
deposit diagnosed by DECT.
Gout can be confirmed by a joint fluid test to reveal urate crystal
deposition in the kidney is the most common and can present as acute uric acid nephropathy, chronic urate nephropathy, or uric acid nephrolithiasis.
Gout (urate crystal
deposition disease) is considered by some to be the most painful and most frequent of all inflammatory joint conditions.
Commenting on how the vitamin may be protecting against gout, the researchers noted that vitamin C may reduce levels of uric acid in the blood thereby preventing the formation of the urate crystal
. This may be achieved by vitamin C having an effect on the reabsorption of uric acid by the kidneys.