tubular

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Related to tubular transport: tubular reabsorption, tubular secretion

tu·bu·lar

(tū'byū-lăr),
Relating to or of the form of a tube or tubule.
Synonym(s): tubuliform

tubular

/tu·bu·lar/ (too´bu-lar)
1. shaped like a tube.
2. of or pertaining to a tubule.

tubular

(to͞o′byə-lər, tyo͞o′-)
adj.
1. Relating to or shaped like a tube: tubular casings; tubular flowers.
2. Consisting of tubes or a tube: a tubular frame.

tu′bu·lar′i·ty (-lăr′ĭ-tē) n.
tu′bu·lar·ly adv.

tubular

See tubule.

tubular

1. pertaining to renal tubules.
2. pertaining to fallopian tube.

tubular backleak
leakage of tubular fluid into the interstitium of the kidney is one of the factors in the pathogenesis of acute renal failure.
tubular maxima
the concentrations of solutes at which the renal tubules are working at full capacity and further increases in concentration will not increase the function. Called also Tm.
tubular necrosis
acute necrosis of the tubular epithelium caused usually by ischemia or exposure to a nephrotoxin; in most cases the patient succumbs to uremia in a few days. See also nephrosis.
tubular proteinuria
failure of the tubules to resorb small molecule proteins excreted by the glomerulus.
tubular reabsorption
reabsorption of solutes from the glomerular filtrate by the tubules, the conservation of protein, glucose and bicarbonate, and the conservation of the water that accompanies them.
tubular transport
refers to all processes which occur with renal tubular fluid during its transport from glomerular space to renal pelvis.
tubular transport maximum
when the tubular transport maximum for a renal tubular solute is exceeded the solute appears in the urine.
References in periodicals archive ?
ROS alter renal medullary oxygen balance by both the enhancement of tubular transport and oxygen consumption and by disrupting nitrovasodilation.
Increased solute delivery to the nephron, augmented tubular transport, and oxygen consumption reflects enhanced GFR and osmotic diuresis [1, 2].
It is tempting to assume that in advanced diabetic nephropathy with reduced numbers of nephrons, oxygen consumption for tubular transport per remnant nephron might be larger, with a greater risk for the development of critical hypoxia.
noted that the administration of antioxidants not only reduced oxygen consumption for tubular transport in the diabetic kidney, but also somewhat improved outer medullary microcirculation [18].
As outlined thoroughly elsewhere [40], adenosinehas a marked influence upon renal parenchymal oxygenation, playing a central role in the tubuloglomerular feedback mechanism, affecting both tubular transport activity and the renal microcirculation.