tubular reabsorption

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The flow of glomerular filtrate from the proximal tubule of the nephron into the peritubular capillaries, which allows the selective passage of certain substances (glucose, proteins, sodium) back into the circulation.

tubular reabsorption

The recovery of needed substances from the contents of the tubule of the nephron into the peritubular capillaries.


1. pertaining to renal tubules.
2. pertaining to fallopian tube.

tubular backleak
leakage of tubular fluid into the interstitium of the kidney is one of the factors in the pathogenesis of acute renal failure.
tubular maxima
the concentrations of solutes at which the renal tubules are working at full capacity and further increases in concentration will not increase the function. Called also Tm.
tubular necrosis
acute necrosis of the tubular epithelium caused usually by ischemia or exposure to a nephrotoxin; in most cases the patient succumbs to uremia in a few days. See also nephrosis.
tubular proteinuria
failure of the tubules to resorb small molecule proteins excreted by the glomerulus.
tubular reabsorption
reabsorption of solutes from the glomerular filtrate by the tubules, the conservation of protein, glucose and bicarbonate, and the conservation of the water that accompanies them.
tubular transport
refers to all processes which occur with renal tubular fluid during its transport from glomerular space to renal pelvis.
tubular transport maximum
when the tubular transport maximum for a renal tubular solute is exceeded the solute appears in the urine.
References in periodicals archive ?
The proposed mechanism of exercise-induced proteinuria involves increased glomerular permeability and exceeding the maximum tubular reabsorption capacity [24,25].
Na]) and tubular handling: fractional proximal tubular reabsorption (FPT[R.
Serum levels, although followed as a useful prognostic marker reflecting tumor burden,[2] may not necessarily parallel urinary levels given that serum concentrations ultimately result from interactions among rates of synthesis, glomerular filtration, and proximal tubular reabsorption.
3,8] Protein loss from the kidney may be caused by changes in glomerular capillary permeability, pore size and charge of basement membrane, tubular reabsorption, or catabolism.
The pyrazinamide suppression test initially demonstrated that this constellation of findings is caused by an enhanced tubular reabsorption of urate rather than by reduced secretion (105).
Lipocalins are small proteins that undergo glomerular filtration and subsequent tubular reabsorption (18), although specific data on FABP4 renal metabolism is lacking.
The increased loss of these proteins in urine is a reflection of the decreased tubular reabsorption capacity.
Because plasma free cortisol is filtered through the glomeruli with partial tubular reabsorption, the amount of free cortisol appearing in the urine is theoretically dependent on the glomerular filtration rate.
As verified by Beckett and Gorrod (16), increased urine flow would be expected to reduce tubular reabsorption of cotinine and enhance elimination, mitigating any dilutional effects.
The restricted tubular reabsorption may lead to the appearance of cTnT and cTnI in urine.