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Relating to, caused by, or characterized by thrombosis.
Farlex Partner Medical Dictionary © Farlex 2012


Relating to, caused by, or characterized by thrombosis.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012

Patient discussion about thrombotic

Q. How can I prevent blood clots? I am 45 years old and am supposed to go on a business trip overseas. The flight itself is 12 hours long and then I have to continue traveling by bus. Could this cause me to have blood clots? If so, how can I prevent it?

A. Always walk as much as you can on the plane. Also, rotate your ankels in circles. Sometimes try to use your ankels and make the alphabet with them. Have fun..

More discussions about thrombotic
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References in periodicals archive ?
Tsai, "Pathophysiology of thrombotic thrombocytopenic purpura," International Journal of Hematology, vol.
Present study was done to determine the serum lipid profile in 60 patients of cerebrovascular disease (44 with thrombotic stroke and 16 with haemorrhagic stroke) with a view to determine lipid abnormalities if any.
Numerous conditions such as acute pancreatitis, connective tissue disorders, autoimmune diseases, pregnancyrelated diseases, and thrombotic microangiopathic diseases can cause Purtscher-like retinopathy.
Hydroxyurea, interferon alfa, and peginterferon alfa, and possibly anagrelide, have been shown to be effective for the prevention of venous thrombotic complications in patients with high-risk ET.
Causes of TMA include thrombotic thrombocytopenic purpura, haemolytic uraemic syndrome, drug-induced TMA, HELLP syndrome, systemic infections and autoimmune disorders amongst many.
Nester, "Syndromes of thrombotic microangiopathy," New England Journal of Medicine, vol.
This is a case of thrombotic thrombocytopenic purpura presented with cerebral venus thrombosis complicated with recurrent generalized seizer.
The biopsy showed evidence of a thrombotic microangiopathic process (Figure 2).
Thrombotic microangiopathy, or TMA, is a condition where a blood clot occurs in capillaries and arterioles due to an endothelial injury.
In September 2018, Akari announced in relation to treatment variables that in the first two patients treated with Coversin as part of a U.K.-named patient program, it had observed a rapid reduction of the markers of complement activation as well as normalization of markers that are elevated in thrombotic microangiopathy (TMA).
Background: The patterns of nonadherence to antiplatelet regimen in stented patients (PARIS) thrombotic risk score are a novel score for predicting the risk of coronary thrombotic events (CTEs) after percutaneous coronary intervention (PCI) with drug-eluting stents.