thrombopoiesis


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Related to thrombopoiesis: leukopoiesis

thrombopoiesis

 [throm″bo-poi-e´sis]
2. thrombocytopoiesis. adj., adj thrombopoiet´ic.

throm·bo·poi·e·sis

(throm'bō-poy-ē'sis),
Precisely, the process of a clot forming in blood, but generally used with reference to the formation of blood platelets (thrombocytes).
[thrombo- + G. poiēsis, a making]

thrombopoiesis

/throm·bo·poi·e·sis/ (-poi-e´sis)
2. thrombocytopoiesis.thrombopoiet´ic

thrombopoiesis

(thrŏm′bō-poi-ē′sĭs)
n.
1. The process of blood clot formation.
2. The formation of blood platelets.

throm′bo·poi·et′ic (-ĕt′ĭk) adj.

throm·bo·poi·e·sis

(throm'bō-poy-ē'sis)
Precisely, the process of a clot forming in blood, but generally used with reference to the formation of blood platelets (thrombocytes).
[thrombo- + G. poiēsis, a making]

thrombopoiesis

1. thrombogenesis.
2. thrombocytopoiesis.
References in periodicals archive ?
The significance of platelets with increased RNA content (reticulated platelets): a measure of the rate of thrombopoiesis.
Therefore, their measurement provides the physician with an assessment of bone marrow thrombopoiesis from a peripheral blood sample in a way that is similar to the assessment of erythropoiesis using reticulocyte counts.
Effects of lithium on thrombopoiesis in patients with low platelet cell counts following chemotherapy or radiotherapy.
Studies on thrombopoiesis I: A factor in normal human plasma required for platelet production; chronic thrombocytopenia due to its deficiency.
The exact mechanisms of thrombopoiesis are still not fully understood.
Rather, the late increase in PMP and decrease in platelet count may be a consequence of altered thrombopoiesis in response to the inflammatory reaction during MI (24).
The strong inverse correlation between thrombopoietin and platelets, as well as the fact that the scatter diagrams (not shown) reveal clear gradients, lends credence to the dioxin influence at the megakaryocyte-to-thrombocyte differentiation stage or earlier in the thrombopoiesis.
Earlier, the pathophysiology of thrombocytopaenia in liver disease was linked to the presence of hypersplenism, but now it has been discovered that thrombopoietin production is dependent on functioning liver cell mass and is responsible for reduced thrombopoiesis and consequently peripheral thrombocytopaenia.
Although no particular mechanism of thrombocytopenia in malaria has been proposed, researchers have reported reduced thrombopoiesis could be a causative factor for thrombocytopenia in the infections of P falciparum and P vivax.
The platelet-vessel wall interaction in experimental atherosclerosis and ischaemic heart disease with special reference to thrombopoiesis.