supraventricular tachycardia


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tachycardia

 [tak″e-kahr´de-ah]
abnormally rapid heart rate, usually taken to be over 100 beats per minute. adj., adj tachycar´diac.
A, Sinus tachycardia; B, Ventricular tachycardia. From Chernecky, 2001.
antidromic circus movement tachycardia a supraventricular tachycardia supported by a reentry circuit that uses the atrioventricular node in the retrograde direction and an accessory pathway in the anterograde direction; this produces a broad QRS rhythm indistinguishable from ventricular tachycardia. Such a tachycardia may also use two accessory pathways (one anterograde and one retrograde) and not involve the AV node at all.
atrial tachycardia a rapid heart rate, between 140 and 250 beats per minute, with the ectopic focus in the atria and with no participation by the atrioventricular node or the sinoatrial node. It is recognizable on the electrocardiogram because the P wave precedes the QRS complex, as opposed to being merged with it or following it. This condition is usually associated with atrioventricular block or digitalis toxicity.
benign ventricular tachycardia tachycardia originating in the ventricles, not associated with structural heart disease or significant hemodynamic symptoms.
bidirectional ventricular tachycardia (bifascicular ventricular tachycardia) a ventricular arrhythmia characterized by heart rates of 90 to 160 beats per minute, alternating right and left axis deviation, ectopic focus that alternates between the anterior superior and posterior inferior fascicles, and a right bundle branch block pattern in lead V1; seen in digitalis toxicity and other conditions.
chaotic atrial tachycardia an ectopic atrial tachycardia due to multifocal activity, characterized by at least three different shapes of P waves on the electrocardiogram; often associated with chronic obstructive lung disease.
circus movement tachycardia (CMT) a reentry circuit that uses an accessory pathway or pathways; there are two subtypes, antidromic and orthodromic circus movement tachycardia.
ectopic tachycardia rapid heart action in response to impulses arising outside the sinoatrial node.
junctional tachycardia rhythm at the rate of 100 to 140 beats per minute that arises in response to impulses originating in the atrioventricular junction, i.e., the atrioventricular node. It is often seen with digitalis toxicity and is due to triggered activity, but it may also be due to altered automaticity. In the case of digitalis toxicity, the term may be used to encompass the entire span of junctional rates with this condition, i.e., approximately 70 to 140 beats per minute.
monomorphic ventricular tachycardia a type that has a uniform beat-to-beat QRS morphology.
nonsustained ventricular tachycardia a type that terminates spontaneously within 30 seconds and does not lead to hemodynamic collapse.
orthodromic circus movement tachycardia a supraventricular tachycardia supported by a reentry circuit that uses the atrioventricular node in the anterograde direction and an accessory pathway in the retrograde direction, producing a narrow QRS complex.
orthostatic tachycardia disproportionate rapidity of the heart rate on arising from a reclining to a standing position.
paroxysmal tachycardia rapid heart action that starts and stops abruptly.
paroxysmal atrial tachycardia paroxysmal supraventricular tachycardia.
paroxysmal supraventricular tachycardia (PSVT) a narrow QRS tachycardia that begins and ends abruptly; it may be terminated with a vagal maneuver. It has two common mechanisms, atrioventricular nodal reentry and circus movement that uses the atrioventricular node anterogradely and an accessory pathway retrogradely. On the electrocardiogram it is characterized by abrupt onset, and mechanisms are differentiated by the relation of the P wave to the QRS complex.
polymorphic ventricular tachycardia a type that has a constantly, and sometimes subtly, changing beat-to-beat QRS configuration.
potentially malignant ventricular tachycardia a type that is not associated with structural heart disease or hemodynamically important cardiac symptoms but is sometimes associated with left ventricular dysfunction.
sinus tachycardia (ST) a rapid rhythm originating in the sinoatrial node with a rate of usually 100 to 160 beats per minute; conduction through the ventricles is normal. During exercise or stress this is normal, but if it occurs during rest it is abnormal.
supraventricular tachycardia a combination of junctional tachycardia and atrial tachycardia.
sustained ventricular tachycardia tachycardia that lasts more than 30 seconds and leads to hemodynamic collapse.
ventricular tachycardia an abnormally rapid ventricular rhythm with aberrant ventricular excitation, characterized by at least three consecutive ventricular complexes of more than 100 beats per minute. It is generated within the ventricle and is most often associated with atrioventricular dissociation.

su·pra·ven·tric·u·lar tach·y·car·di·a (SVT),

rapid heart rate due to a pacemaker anywhere above the ventricular level, that is, sinus node, atrium, atrioventricular junction. The QRS complexes are always narrow unless there is rate-related aberrancy or preexisting intraventricular conduction delay.

supraventricular tachycardia

Paroxysmal atrial tachycardia Cardiology An arrhythmia initiated by a premature atrial beat in the AV node, SA node, or bundle to the point of bifurcation; once the ventricle contracts, an echo atrial beat is stimulated via a retrograde tract, resulting in a reverberating re-entry phenomenon, resulting in an atrial rate of 180-300 beats/min Management Simple vagal stimulation–eg, carotid sinus massage, ice bag, breath-holding; if intense, cardioversion or digoxin therapy

supraventricular tachycardia

Episodes of abnormally fast heart-rate lasting for hours or days. The rate may be as high as 300 beats per minute but is usually between 140 and 180. It is caused by fast spontaneous impulses, arising in the upper chambers of the heart, that over-ride the natural pacemaker. There may be chest pain, breathlessness, consciousness of the heart action (PALPITATIONS) and faintness. Attacks may be stopped by the VALSALVA MANOEUVRE or by the use of antiarrhythmic drugs. Sometimes electrical cardioversion is necessary.

Supraventricular tachycardia

A fast heart beat that originates above the ventricles.

Patient discussion about supraventricular tachycardia

Q. SVT and AF, Hearts that go fast to slow or any others probs with the beats of any kind and Ablation of hearts I have had Ablation done once and I am still having passing out spells and still on 50mg toprol 2 times a day till two days ago, now I am on 150 to 200 aday again. Its not the first time I have had to up meds. I had ablation down 4/22/05. I can breath better now but but it didnt take it away as you can tell. Now Dr Leonardie would like to do it again . This is the big ????! Will it or can it work 100% this time, or will it hit and miss some again???? MTT

A. Well I can understand the frustration of having to go through this procedure yet another time. There are no guarantees in medicine. You should follow your doctor's advice, as another proceudre might be more helpful than the last one. However you should keep in mind that nothing is for sure.

More discussions about supraventricular tachycardia
References in periodicals archive ?
Which patient should be referred to an electrophysiologist: supraventricular tachycardia. Heart.
Contemporary management of paroxysmal supraventricular tachycardia. Circulation 2003;107:1096-9.
Oral, "Value of ST-segment depression during paroxysmal supraventricular tachycardia in the diagnosis of coronary artery disease," The American Journal of Cardiology, vol.
Ramlakhan, "Handstands: A treatment for supraventricular tachycardia?" Archives of Disease in Childhood, vol.
Adenosine versus intravenous calcium channel antagonists for the treatment of supraventricular tachycardia in adults.
A wide complex tachycardia has a QRS duration >120 ms and may be caused by one of the following mechanisms: (i) ventricular tachycardia, which must be the default diagnosis in any patient with a wide complex tachycardia; (ii) supraventricular tachycardia with a right or left bundle branch block or an intraventricular conduction abnormality; (iii) pre-excited tachycardia (Wolff-Parkinson-White syndrome) over an accessory pathway; and (iv) pacemaker tachycardia.
clinical trials by their phase, trial status, prominence of the sponsors and also provides briefing pertaining to the number of trials for the key drugs for treating Supraventricular Tachycardia. This report is built using data and information sourced from proprietary databases, primary and secondary research and in-house analysis by GlobalData's team of industry experts.
According to the ICD no VT was detected in the follow-up period, and only paroxysmal supraventricular tachycardia was detected.
Medical treatment of supraventricular tachycardia often involves regular intake of drugs for several years.
Adenosine and calcium channel blockers are the intravenous drugs of choice for termination of paroxysmal supraventricular tachycardia.5,6 Medical treatment of SVT with antiarrhythmic drugs has limited efficacy.
The incidence of multifocal atrial tachycardia (MAT) in infants and children is very low, accounting for approximately 1% of supraventricular tachycardia (SVT) substrates.
Edward Ives was born with just a five percent chance of survival due to a condition called supraventricular tachycardia (SVT), which causes the heart to race dangerously fast.