Lying dorsal to the anterior or posterior cardinal veins in the embryo.
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LSIVC is relatively less prevalent (0.2%-0.5%) than PLSVC (3) and develops due to the persistence of the left supracardinal vein(4).
The inferior vena cava (IVC) regularly develops according to a complex process which involves three pairs of fetal veins: the posterior cardinal veins, subcardinal veins, and supracardinal veins [5].
During the fifth to the eighth week of embryonic period, the IVC develops as a composite structure via chronological events of formation, regression, anastomosis and substitution of three symmetrical sequential pairs of veins, the posterior cardinal, subcardinal and supracardinal (Artico et al., 2004).
Lastly, the supracardinal veins appear with the right system persisting to form the infrarenal segment of IVC and the left system regressing.
No embryologic remnants of the supracardinal veins are noted on the right side of the aorta.
Regardless of the theory, we find that the failure of the supracardinal vein to persist as IVC is a common point [17].
La porcion infrarrenal de la vena cava inferior se forma a partir de la anastomosis de las venas subcardinalsupracardinal con la supracardinal derecha [1,2] (figura 4).
It involves a complex process comprising 3-pairs of embryogenic veins: posterior cardinal (iliac and confluent), subcardinal (renal and hepatic) and supracardinal (prerenal).
In the thoracic region, the supracardinal veins give rise to the azygos and hemiazygos veins.
The post-hepatic IVC develops between the sixth and eighth weeks of embryonic life as a composite structure formed from the continuous appearance and regression of three paired embryonic veins, the posterior cardinal, the subcardinal, and the supracardinal veins.
(1) Failure of the left caudal supracardinal vein to regress results in persistent communication between the left common iliac vein and the left renal vein.