sucrase-isomaltase


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sucrase-isomaltase

(1) Oligo-1,6-glucosidase, EC 3.2.1.10.  
(2) Sucrose alpha-glucosidase, EC 3.2.1.48.
References in periodicals archive ?
Enzyme-substitution therapy with the yeast Saccharomyces cerevisiae in congenital sucrase-isomaltase deficiency.
com)-- Popular morning show The Balancing Act[R] will feature the rare disorder of Congenital Sucrase-Isomaltase Deficiency as part of the “Behind the Mystery: Rare & Genetic Diseases” series on the show airing on Friday, July 26th, at 7:00 a.
These glucosidases are comprised of two enterocyte membrane bond complexes of N- and C-terminals each, as well as of maltase-glucoamylase (MGAM) and sucrase-isomaltase, making them the ultimate step in producing dietary glucose.
Sucraid oral solution is an enzyme replacement therapy (ERT) for the treatment of genetically determined sucrase deficiency, which is part of congenital sucrase-isomaltase deficiency (CSID), a rare, chronic disease.
Glucagon-like peptide 2 enhances maltase-glucoamylase and sucrase-isomaltase gene expression and activity in parenterally fed premature neonatal piglets.
Sucraid provides drug replacement therapy for patients that have been diagnosed with a congenital (inherited) disease called Congenital Sucrase-Isomaltase Deficiency (CSID).
Sucrase-isomaltase is an independent prognostic marker for colorectal carcinoma.
The purchaser will continue to serve patients who use Sucraid to treat sucrase-isomaltase deficiency, a rare metabolic disorder.
Orphan Europe works with the following diseases: acute porphyria, congenital sucrase-isomaltase deficiency, homocystinuria, hyperammonaemia due to NAGS deficiency, nephropathic cystinosis, patent ductus arteriosus, severe combined immunodeficiency - adenosine deaminase deficiency, vitamin E deficiency in chronic cholestasis and Wilson's disease.
It has been proposed that sucrase-isomaltase contributes approximately 80% of the mucosal maltase activity and maltase-glucoamylase contributes 20% (Galand, 1989).
Patients with rare hereditary problems of fructose intolerance, glucose-galactose malabsorption or sucrase-isomaltase insufficiency should not take this medicine.
Sucrase-isomaltase and hexose transporter gene expression are coordinately enhanced by dietary fructose in rat jejunum.