QRS duration and dispersion as well as temporal changes in these parameters, left ventricle ejection fraction and subpulmonary
ventricular functions were predictors of sudden cardiac death in ACHD patients.
The subaortic conus is absorbed and the subpulmonary
conus growth brings the pulmonary artery superiorly to the left of aorta so that the right ventricular outflow tract crosses the aorta anteriorly.
ventricular dysfunction, pulmonary regurgitation, or both, which conferred a 3.4-fold independent increased risk for a late event.
infundibulum) with atresia of the pulmonary valve, a large VSD, and overriding of the aorta.
The ECHO showed a malalignment VSD (as found after birth), but now there is evidence of subpulmonary
stenosis that was not present on her newborn studies.
valve obstruction progressed in one woman and a new obstruction appeared in another.
Continuous wave Doppler revealed 200 mmHg pressure gradient across the stenotic pulmonary valve associated with secondary hypertrophic subpulmonary
The Amplatzer device was seen to be wedged in the subpulmonary
infundibulum, lying against the leaflets of the pulmonary valve (Fig.
In this case, we considered subpulmonary
fibromuscular tissue, which causes LVOTO, is correctable with resection.
The other associated cardiac anomalies include left coronary artery originating from the pulmonary trunk, coronary arterioventricular fistulae, right coronnary artery atresia, pulmonar artery atresia, subpulmonary
stenosis, anomalous pulmonary venous return, patent ductus arteriosus, tetralogy of Fallot, transposition of the great arteries, dextrocardia and aortic atresia (8,9).