subendothelium

sub·en·do·the·li·um

(sŭb'en-dō-thē'lē-ŭm),
The connective tissue between the endothelium and inner elastic membrane in the intima of arteries.

sub·en·do·the·li·um

(sŭb'en-dō-thē'lē-ŭm)
The connective tissue between the endothelium and inner elastic membrane in the intima of arteries.

subendothelial

, subendothelium (sŭb″ĕn-dō-thē′lē-ăl) (sŭb″ĕn-dō-thē′lē-ŭm) [″ + Gr. endon, within, + thele, nipple]
Beneath the endothelium.
References in periodicals archive ?
Pathologic diagnosis was based on the presence of all 3 of the following histologic signs: widening of subendothelium with myxoid degeneration, intraluminal thrombus formation, and fibrinoid necrosis of terminal arterioles.
The other possible mechanisms are chemical arteritis and tissue destruction of endothelium, subendothelium, or muscle layers of vessel (1, 6, 7).
22) Anemia could further worsen the bleeding disorder because red cells release ADP and facilitate platelets contacting with subendothelium at the damage site.
Migration of monocytes into the subendothelium is the initial step of inflammation and atherosclerotic plaque formation, which plays a key role in the atherosclerotic plaque formation.
Nevertheless, one should not ignore any damage to the endothelium and subendothelium by pressure of convex ends of intracoronary shunts (especially when too big shuts are used).
Atherosclerosis begins as a disease of the subendothelium and the vessel wall in direct response to the foods and inflammatory milieu that we create.
The HDL cholesterol is a lipoprotein that carries out the reverse cholesterol transport, removing the excess of free cholesterol not only from cell membranes but also from the subendothelium and transporting it to the liver to be degraded (Shils et al.
This condition enhances fatty streaks formation because lipid migration into the subendothelium is greater than its removal from the arterial wall.
Upon vascular damage subendothelium macromolecules such as collagen thrombin ADP serotonin (5-HT) and thromboxane A2 (TxA2) are exposed or secreted at the site of damage and platelets adhere to exposed subendothelium which are activated and leads to hemostasis (Hubbard et al.
In 13 cm CVRL foetus the thin layer of subendothelium was observed with large number of differentiating Purkinje fibres.
Von Willebrand Factor (VWF) is a polymeric plasma glycoprotein comprising multiple 250-kDa subunits that facilitates platelet adhesion to the vessel wall by linking platelet membrane receptors to the subendothelium and is the cornerstone of platelet--endothelial interaction.