Although the reasons for the absence of abnormal Q waves are not fully understood, they are associated with subendocardial
rather than transmural infarction, inferior rather than anterior infarcts, and smaller rather than larger infarcts in adults.
This sequence is also useful for distinguishing between ischemic cardiomyopathy with the presence of segmental, subendocardial
LGE or transmural LGE from a non-ischemic process, such as with myocarditis or Takotsubo cardiomyopathy in patients presenting with acute chest pain and elevated cardiac enzymes.
In a focally extensive area of the subendocardial
myocardium, cardiomyocytes were replaced by an eosinophilic fibrillar material that stained blue with trichrome stain (fibrosis/myocardial infarction).
Further histopathologic examination of the raised left atrial lesion demonstrated the characteristic features seen in a MacCallum plaque, or mitral regurgitation jet lesion, of bland, loose subendocardial
Cardiac MRI following one of these episodes confirmed an acute subendocardial
Burton, Korsheed, Grundy, and McIntyre (2008) found a higher incidence of premature ventricular contractions in patients who developed regional wall motion abnormalities during HD, noting that this may be related to demand ischemia--Selby and McIntyre (2007) stated that increased arterial stiffness adversely effects myocardial perfusion, reducing the ischemic threshold and increasing the risk of reduced subendocardial
Other theories which have been suggested to explain are: impaired coronary vascular reactivity3 subendocardial
hypoperfusion detected by 3T MRI3 micro vascular dysfunction13 metabolic abnormalities-such as net spasm vasculitis and misinterpreta-tion of coronary angiograms.
Fatal acetaminophen poisoning with evidence of subendocardial
necrosis of the heart.
26] have shown that there is a cyclic release of NO in the heart during heart rate, mainly in subendocardial
zone, which reaches its peak during ventricular relaxation and early rapid chamber filling phase.
The heart had multifocal areas of hemorrhage in the subepicardial and subendocardial
First, most patients with renal insufficiency demonstrate LV hypertrophy and subsequent subendocardial
ischemia secondary to arterial hypertension even prior to ESRD requiring dialysis.
A clinical study of Capros from Natreon found that supplementation resulted in a significant decrease in the indices of arterial stiffness (augmentation index and radial and aortic blood pressures) and a significant increase in subendocardial
viability ratio (SEVR), an index of myocardial perfusion, when subjected to a cold pressor test.