stasis syndrome


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stasis

 [sta´sis]
a stoppage or diminution of flow, as of blood or other body fluid, or of intestinal contents.
stasis syndrome overgrowth of bacteria within the small intestine resulting from a variety of conditions causing stasis, particularly disturbances to intestinal motility or decreased acid secretion, but also structural abnormalities such as diverticula, fistulae between the colon and upper bowel, or chronic obstruction; it is characterized by malabsorption of vitamin B12, steatorrhea, and anemia.
venous stasis cessation or impairment of venous flow, such as with venous insufficiency; see also stasis ulcer. Called also phlebostasis and venostasis.

stasis syndrome

overgrowth of bacteria in the small intestine resulting from a variety of conditions causing stasis, particularly disturbances to intestinal motility or decreased acid secretion but also structural abnormalities such as diverticula, fistulae between the colon and upper bowel, or chronic obstruction. It is characterized by malabsorption of vitamin B12, steatorrhea, and anemia. Also called bacterial overgrowth syndrome, blind loop syndrome.
References in periodicals archive ?
16 There are few studies to observe the relationship between E2 and lipid or inflammtory factors between the healthy menopausal women menopausal women with blood stasis syndrome and menopausal women with non-blood stasis syndrome.
A number of studies have confirmed that compared with the non-blood stasis syndrome group and healthy control group the levels of hs-CRP sICAM-1 sVCAM-1 in coronary blood stasis syndrome group are significantly increased.
Further subgroup analysis found that serum sICAM-1 levels were incremental increased in the healthy control group non-BSS group and BSS group and sICAM-1 level in BSS group was significantly higher than the other two groups which indicated that during the pathological process of atherosclerosis patients with coronary blood stasis syndrome were subjected to more severe chronic inflammatory reactions than the patients with non-blood stasis syndrome and E2 levels and sICAM-1 were negatively correlated.