small vessel vasculitis

Internal medicine Vasculitis affecting vessels smaller than arteries–eg, arterioles, venules, and capillaries Clinical Palpable purpura, nodules, ulceration, urticaria; 30-50% involve GI tract and are accompanied by fever, neuritis, glomerulonephritis Examples Churg-Strauss syndrome, cutaneous leukocytoclastic angiitis, essential cryoglobulinemic vasculitis, Henoch-Schönlein purpura, microscopic polyangiitis, Wegener's granulomatosis, etc. See Systemic vasculitis.

Mentioned in ?

References in periodicals archive ?

Infections linked to causing small vessel vasculitis include streptococcal and staphylococcal infections, tuberculosis, leprosy, hepatitis B, hepatitis C, HIV, and infective endocarditis.

Rare Association of Leukocytoclastic Vasculitis in Visceral Leishmaniaisis

Endothelial dysfunction, increased cardiovascular events (CVE), and accelerated atherosclerosis have been described in patients with systemic autoimmune diseases such as large and small vessel vasculitis and collagen vascular disease (1, 2).

Higher levels of SDMA and not ADMA are associated with poorer survival of trial patients with systemic ANCA-associated vasculitis

In cases of small vessel vasculitis the magnetic resonance angiography findings are usually normal22.

CLINICAL SPECTRUM AND NEURO IMAGING FINDINGS OF PATIENTS WITH CEREBRAL ARTERIOPATHY

Imai et al., "Infective endocarditis by Bartonella quintana masquerading as antineutrophil cytoplasmic antibody-associated small vessel vasculitis," Cardiology, vol.

Bartonella Endocarditis Mimicking Crescentic Glomerulonephritis with PR3-ANCA Positivity

ANCA is regarded as a marker of small vessel vasculitis. However, some patients who test positive for ANCA exhibit infections (such as tuberculosis), inflammatory bowel diseases, other autoimmune disorders, and malignancies [6-10].

Nasal Septal Perforation in Propylthiouracil-Induced Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis

ANCA is strongly associated with three types of small vessel vasculitis: microscopic polyangiitis (MPA), granulomatosis with polyangiitis (GPA), and eosinophil granulomatosis with polyangiitis (EGPA) [3].

Monocyte Chemoattractant Protein-1 in Antineutrophil Cytoplasmic Autoantibody-Associated Vasculitis: Biomarker Potential and Association with Polymorphisms in the MCP-1 and the CC Chemokine Receptor-2 Gene

Henoch-Schonlein purpura (HSP) is the most common form of systemic small vessel vasculitis involving the skin, joints, kidneys, and gastrointestinal tract in childhood.

Cerebral Vasculitis in Henoch-Schonlein Purpura: A Case Report

Introduction: IgA vasculitis is a systemic small vessel vasculitis accompanied by gastrointestinal tract (GIT) and renal involvement.

CASE REPORT: IGA VASCULITIS WITH RESISTANT GASTROINTESTINAL FINDINGS

(10) The previous consensus nomenclature system (11) was developed prior to widespread ANCA serologic testing and did not differentiate sufficiently the different forms of small vessel vasculitis. One of the main areas of revision included the description of 2 pathophysiologically different groups of small vessel vasculitis including immune complex vasculitis and AAV (Table 1).

Pathology of Antineutrophil Cytoplasmic Antibody--Associated Pulmonary and Renal Disease

The renal manifestations are typically a rapidly progressive crescentic necrotizing glomerulonephritis that is caused by small vessel vasculitis within glomeruli [1].

Granulomatosis with Polyangiitis with Myocarditis and Ventricular Tachycardia

E, Small vessel vasculitis with neutrophilic inflammation and leukocytoclasia (H&E, magnification 100x).

Cocaine/levamisole-induced systemic vasculitis with retiform purpura and pauci-immune glomerulonephritis