sickle cell syndrome

sickle cell syndrome

Patient discussion about sickle cell syndrome

Q. Please precribe for me the possible medicine (treatment) for sickle cells. Secondly, my boy lost hearing at 4 1- I need to know how sickle cells can be treated. 2- My boy just surprisingly lost his abillity to hear anything at the age of 4.

A. wow...you are going through some hard times...it's the hardest thing in the world seeing your children in pain. loosing his hearing could be caused by clots that were formed and destroyed the ear nerve. but it's unlikely it happened in both ears...so i would check it out. and about treatment- there are a variety of treatments, so i found a web site with them all. and even some that are still in research: http://www.nhlbi.nih.gov/health/dci/Diseases/Sca/SCA_Treatments.html

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References in periodicals archive ?
Psychosocial and family functioning in children with sickle cell syndrome and their mothers.
Predictors of psychosocial and cognitive adaptation in children with sickle cell syndromes.
Red cell deformability and the effect of piracetam in sickle cell syndrome,.
Our findings suggest that family variables play an important role in the depressive symptoms of children with sickle cell syndrome.
Risk-resistance-adaptation model for pediatric chronic illness: Sickle cell syndrome as an example.
Increased RDW is associated with iron deficiency anaemia, megaloblastic anaemia (folate and cobalamin deficiency), haemolytic anaemia, recent blood transfusion, hereditary spherocytosis and sickle cell syndromes [33,34].
In sickle cell syndromes, the primary event is intra-erythrocytic haemoglobin precipitation (gelation), with resultant formation of tactoids, which deforms the discoid red cell to sickle or crescent morphology [36].
Thrombotic microangiopathy in patients with sickle cell syndromes, especially intracardiac thrombi without underlying structural diseases or atrial arrhythmias, is a rare complication.
Most patients with sickle cell syndromes suffer from hemolytic anemia with hematocrits from 15-30% and significant reticulocytosis.
After the rediagnosis, the patient was started on hydroxyurea (1000 mg/day) in accordance with recent NIH consensus documents recommending hydroxy urea treatment in several sickle cell syndromes, including S/C and S/[O.
The clinical severity of thalassemia major and the sickle cell syndromes makes them priority genetic diseases for prevention programs involving population screening for heterozygotes and for optional prenatal diagnosis for carrier couples.