sensorimotor polyneuropathy

sensorimotor polyneuropathy

Neurology A peripheral neuropathy characterized by ↓ movement or sensation, due to nerve damage; SP is a systemic process that damages nerves, with loss of myelin, which slows neural conduction or damages neurons, especially the axon, blocking conduction at the point of damage–pressure on nerves, inflammation, ↓ blood flow, connective tissue disorders, idiopathic, alcoholic neuropathy, diabetic neuropathy, chronic inflammatory neuropathy, Guillain-Barre syndrome, and drug-induced neuropathy. See Neuropathy.
References in periodicals archive ?
3,6) Deficiency of vitamin B12, diabetes mellitus type I and type II, colchicine, and alcohol use may cause sensorimotor polyneuropathy in distal extremities.
Tkac I, Bril V: Glycemic control is related to the electrophysiologic severity of diabetic peripheral sensorimotor polyneuropathy.
Glycemic control is related to the morphological severity of diabetic sensorimotor polyneuropathy.
Distal symmetric sensorimotor polyneuropathy is the most common of all diabetic peripheral neuropathies, affecting 16%-50% of all patients with diabetes.
In summary, the electromyographic examination revealed abnormalities most consistent with a primary axonloss sensorimotor polyneuropathy.
Electromyography and nerve conduction studies demonstrated abnormalities most consistent with a primary axon loss sensorimotor polyneuropathy.
Validation of a Novel Point-of-Care Nerve Conduction Device for the Detection of Diabetic Sensorimotor Polyneuropathy.
The most common lesion is peripheral neuropathy, including entrapment neuropathy, distal axonal predominantly sensory polyneuropathy, mononeuropathy or multiple mononeuropathy, as well as fulminant sensorimotor polyneuropathy (1).
In the RA group, electrophysiologically diagnosed neuropathy was detected in 20 (36%) patients: 3 (5%) had sensorimotor polyneuropathy, 7 (13%) had low sural sensory CV or absence of sensory action potentials (SAP), 2 (4%) had carpal tunnel syndrome (CTS), 6 (11%) had low amplitude peroneal compound muscle action potentials, 1 (2%) had low amplitude peroneal compound muscle action potential and low ulnar sensory nerve conduction, and 1 (2%) had low sural and ulnar sensory nerve conduction.
Respiratory muscles may be depressed, accompanied by an axonal sensorimotor polyneuropathy.
Multi-Site Testing with a Point-of-Care Nerve Conduction Device Can Be Used in an Algorithm to Diagnose Diabetic Sensorimotor Polyneuropathy.
This automated device represents an alternative to conventional nerve conduction studies for the diagnosis of diabetic sensorimotor polyneuropathy.