However over the period of time recommendations by Dhillon and Dhillon (2009) were adopted by farmers like use of gypsum to soils which reduces absorption of Selenium in plants, use of organic manure also reducing Selenium toxicity, changing the cropping sequence with recommendation of crops with less uptake of Selenium and increasing depth of water (deep boring) in affected areas as well as avoiding the feeding of crops grown on seleniferous soils markedly reduced the uptake of Selenium by crops raised on seleniferous soils, It was considered that adoption of these recommendations might have led to decreased incidence of clinical cases and severity of signs thus reducing the effect of chronic selenosis on animal system.
Thereby, indicating that mean Hb, PCV, TEC and TLC in selenotic animals were within normal range and therefore cannot be used in diagnosis and assessment of prognosis of chronic selenosis (Table 1).
Common clinical manifestations of chronic selenosis are rough starry coat, horizontal cracks followed by elongation of hooves and horns leads to sloughing of hooves and avulsion of horns associated with lameness, decline in production and reproduction.
Thus, hair Se was better indicator of chronic selenosis as compared to plasma Selenium concentration.
Tissue selenium as an index of selenosis in buffaloes.
Environmental Protection Agency 2002) are based mostly on reports of chronic selenosis in a Chinese population with excessive Se exposure, resulting from high Se in crops fertilized with coal ash highly rich in Se (Yang et al.
2004) pointed out that there are no recorded signs of selenosis in Greenland populations and that their high Se intake may be tolerated at higher levels.