Effects of selenium deficiency
on testicular morphology and function in rats.
(4) While a deficiency in selenium will not lead to specific symptoms or clinical illness in most people, selenium deficiency
has been identified as a public health concern in certain populations, and coordinated efforts have been made in the past to prevent selenium deficiency
in these populations.
usually results in growth and of activity of enzyme GPx reductions (NRC, 2011).
Protein oxidation is known to be related to selenium deficiency
(Moskovitz and Stadtman, 2003).
Previous trials have reported reduction of TPOAb with selenium after six months and we know that selenium deficiency
leads to glutathione peroxidase inactivity, which may induce oxidative harm for thyroid cells leading to thyroid damage and fibrosis.
Prolonged dietary selenium deficiency
or excess does not globally affect selenoprotein gene expression and/or protein production in various tissues of pigs.
is common in ruminants, mainly due to insufficient dietary levels or the formation of insoluble compounds in the rumen that inhibit their absorption (SPEARS, 2003).
In this way, selenium deficiency
can result in both production of T3 and elimination of [H.sub.2][O.sub.2] inefficient, moreover it contributes to initiation of oxidative damage, fibrosis and impaired thyroid tissue repair.
Unfortunately, as has been stated in many countries across the world, human food ingredients can contain low levels of selenium and selenium deficiency
in human nutrition has become a global phenomenon [17-19].
(7,8) According to previous studies, zinc and selenium deficiency
is common in cirrhosis and can affect nitrogen metabolism.
has been associated with cognitive decline in the elderly (Akbaraly et al.
Effect of dietary selenium deficiency
on the in vitro fertilizing ability of mice spermatozoa.