secondary gout

sec·on·dar·y gout

gout resulting from increased serum uric acid levels as a result of an antecedent disease, such as a proliferative disease of the blood and bone marrow, lead poisoning, or prolonged chronic renal failure (on dialysis).

sec·on·dar·y gout

(sek'ŏn-dar-ē gowt)
Gout resulting from increased serum uric acid levels as a result of an antecedent disease, such as a proliferative disease of the blood and bone marrow, lead poisoning, or prolonged chronic renal failure (on dialysis).
References in periodicals archive ?
Age (mean [+ or -] SD) 63.7 [+ or -] 12.6 Gender n (%) Male 4318 (73%) Female 1561 (26%) No data available 69 (1%) Gout flares/year 3 [+ or -] 2.3 (mean [+ or -] SD) Number of patients with n (%) Primary gout 4 008 (67%) Secondary gout 1278 (21%) No data available 662 (11%) Number of patients with n (%) Gout in the big toe joint 4138(70%) Tophi 540 (9%) Joint damage/changes * 1077 (18%) Number1 of patients with n (%) concomitant disease With at least one 4489 (75.5%) Hypertension 4102 (69%) Hyperlipidemia 2557 (43%) Diabetes mellitus 1902 (32%) Impaired renal function 976 (16.4%) Thyroid dysfunction 431 (7.3%) Depressions 418 (7%) History of kidney stones 195 (3.3%) Other 854 (14.4%) (1) Percentual reference to number of total collective n = 5948.
Urate lowering therapy with febuxostat in daily practice--a multicentre, open-label, prospective observational study
Since no underlying disease can be identified in the majority of cases, secondary gout is quite rare (13).
Coexisting ankylosing spondylitis and gouty arthritis/Ankilozan spondilit ve gut artriti birlikteligi
Primary gout is caused by an increase in uric acid production, while secondary gout is caused by either a decrease in urinary uric acid excretion or an overproduction of purine secondary to increased cell turnover (e.g., tumor lysis).
Gout
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