secondary gout

sec·on·dar·y gout

gout resulting from increased serum uric acid levels as a result of an antecedent disease, such as a proliferative disease of the blood and bone marrow, lead poisoning, or prolonged chronic renal failure (on dialysis).

sec·on·dar·y gout

(sek'ŏn-dar-ē gowt)
Gout resulting from increased serum uric acid levels as a result of an antecedent disease, such as a proliferative disease of the blood and bone marrow, lead poisoning, or prolonged chronic renal failure (on dialysis).
References in periodicals archive ?
Age (mean [+ or -] SD) 63.7 [+ or -] 12.6 Gender n (%) Male 4318 (73%) Female 1561 (26%) No data available 69 (1%) Gout flares/year 3 [+ or -] 2.3 (mean [+ or -] SD) Number of patients with n (%) Primary gout 4 008 (67%) Secondary gout 1278 (21%) No data available 662 (11%) Number of patients with n (%) Gout in the big toe joint 4138(70%) Tophi 540 (9%) Joint damage/changes * 1077 (18%) Number1 of patients with n (%) concomitant disease With at least one 4489 (75.5%) Hypertension 4102 (69%) Hyperlipidemia 2557 (43%) Diabetes mellitus 1902 (32%) Impaired renal function 976 (16.4%) Thyroid dysfunction 431 (7.3%) Depressions 418 (7%) History of kidney stones 195 (3.3%) Other 854 (14.4%) (1) Percentual reference to number of total collective n = 5948.
Since no underlying disease can be identified in the majority of cases, secondary gout is quite rare (13).
Primary gout is caused by an increase in uric acid production, while secondary gout is caused by either a decrease in urinary uric acid excretion or an overproduction of purine secondary to increased cell turnover (e.g., tumor lysis).