renal tubule


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tubule

 [too´būl]
a small tube; especially, a minute canal found in various structures or organs of the body. adj., adj tu´bular.
collecting t's the terminal channels of the nephrons, which open on the summits of the renal pyramids in the renal papillae.
convoluted t's channels that follow a tortuous course; there are convoluted renal tubules and convoluted seminiferous tubules.
dentinal t's the tubular structures of the teeth.
galactophorous t's small channels for the passage of milk from the secreting cells in the mammary gland.
Henle's t's the straight ascending and descending portions of a renal tubule forming Henle's loop.
lactiferous t's galactophorous tubules.
mesonephric t's the tubules comprising the mesonephros, or temporary kidney, of amniotes.
metanephric t's the tubules comprising the permanent kidney of amniotes.
renal tubule the minute canals composing the substance of the kidney and secreting, collecting and conducting the urine; in the glomerulus they have a basement membrane and epithelial lining.
seminiferous t's the tubules of the testis, in which spermatozoa develop and through which they leave the gland.
uriniferous t's renal tubules; channels for the passage of urine.

renal tubule

The part of a nephron through which renal filtrate from the renal corpuscle flows and is changed to urine by reabsorption and secretion. The parts, in order, are the proximal convoluted tubule, the loop of Henle, the distal convoluted tubule, and collecting tubule.
See: kidney for illus; nephron
See also: tubule
References in periodicals archive ?
Renal Tubules Cloudy Swelling: The epithelial lining of the renal cell of rats subjected to Ti[O.sub.2] NPs (252 mg/kg or more for 24 h or more) showed cloudy swelling (Fig.
The volume of fluid entering and travelling along the renal tubules is not sufficient to support tubular cell survival and an acute kidney injury occurs.
The morphology of the renal tubules was also affected as reflected with numerous swollen mitochondria with a crista disarrangement and partial cristolysis within tubular epithelium (Figure 7(c)).
Urinary excretion of these tubular markers could, therefore, be useful for assessing an initial malfunction or damage of the renal tubules in the early stage of potentially progressive DN.
Blood pH and urinary oxalate and [[alpha].sub.1]-microglobulin (a biomarker of renal tubule damage) excretion were monitored for 48 h.
Increased levels of these two markers in renal tubules usually indicate uptake and transport problems in the tubules.
CaOx crystals generally form in the renal tubules. Exposure of renal epithelial cells to CaOx crystals results in the increased synthesis of osteopontin, bikunin, heparan sulphate, monocyte chemoattractant protein (MCP)-l, and prostaglandin (PG) E2, which are known to participate in inflammatory processes and in extracellular matrix production.
Because dehydrated patients are at highest risk of developing CIN due to a higher concentration of toxic substances in the renal tubule, volume expansion is the mainstay of CIN prophylaxis.
In male rats, there were marginal, nonsignificant increases in the incidences of renal tubule adenomas (with one carcinoma at the highest dose) and hepatocellular adenomas, but these findings were considered to be equivocal.
PTH and calcitriol also act on bone to mobilise calcium, and in the distal renal tubule, to stimulate calcium reabsorption.
Previous research suggested that lead may injure the renal tubule cells.

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