renal cortex

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Related to renal cortex: renal pelvis, Renal medulla


 [kor´teks] (pl. cor´tices) (L.)
the outer layer of an organ or other structure, as distinguished from its inner substance or medulla. adj., adj cor´tical.
adrenal cortex (cortex of adrenal gland) the outer, firm layer comprising the larger part of the adrenal gland; it secretes mineralocorticoids, androgens, and glucocorticoids.
cerebellar cortex the superficial gray matter of the cerebellum.
cerebral cortex (cortex cerebra´lis) the convoluted layer of gray matter covering each cerebral hemisphere. See also brain.
renal cortex the granular outer layer of the kidney, composed mainly of glomeruli and convoluted tubules, extending in columns between the pyramids that constitute the renal medulla.
striate cortex part of the occipital lobe that receives the fibers of the optic radiation and serves as the primary receiving area for vision. Called also first visual area.
visual cortex the area of the occipital lobe of the cerebral cortex concerned with vision; the striate cortex is also called the first visual area, and the adjacent second and third visual areas serve as its association areas.
Miller-Keane Encyclopedia and Dictionary of Medicine, Nursing, and Allied Health, Seventh Edition. © 2003 by Saunders, an imprint of Elsevier, Inc. All rights reserved.

re·nal cor·tex

the part of the kidney consisting of renal lobules in the outer zone beneath the capsule and the lobules of the renal columns that are extensions inward between the pyramids; contains the renal corpuscles, medullary rays, and proximal and distal convoluted tubules.
Synonym(s): cortex renalis [TA]
Farlex Partner Medical Dictionary © Farlex 2012

re·nal cor·tex

(rē'năl kōr'teks) [TA]
The part of the kidney consisting of renal lobules in the outer zone beneath the capsule and the lobules of the renal columns that are extensions inward between the pyramids; contains the renal corpuscles and the proximal and distal convoluted tubules.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012
References in periodicals archive ?
As shown in Figure 5, Trxl protein expression (c, d) and mRNA level (e) were decreased in renal cortex following CIN induction (p < 0.01 CON versus CIN).
Bai et al., "Effect of chinese herbs for stasis removing and collaterals dredging upon angiotensin-converting enzyme 2-angiotensin-(1-7)-mas axis in the renal cortex of diabetic nephropathy rats," Chinese Journal of Integrated Traditional and Western Medicine, vol.
The prominent column of Bertin is in continuity with the renal cortex and manifests the same enhancement of the renal parenchyma in all contrastographic phases.
The mRNA Expression of VEGF, Nephrin, and Type IV Collagen in the Renal Cortex. The renal VEGF mRNA expression, according to quantitative RT-PCR, decreased significantly in the taurine-treated diabetic group compared to the diabetic control group (Figure 4(a)).
Analysis of subcellular fractions of the renal cortex revealed cadmium migration from the soluble cytoplasmic fraction (where metallothionein is located) to the mitochondrial fraction (where ATP is maximal) and to the microsomal fraction (where Na-K-ATPase activity is maximal) at the time of appearance of Fanconi syndrome, with reversal after cadmium injection discontinuation.
As expected, PEPCK was exclusively detected in the renal cortex and outer medulla from WT, NaW-WT, IRS2-KO, and NaW-IRS2-KO mice, where proximal tubules are organized (Figures 2(a)-2(d), resp.).
A 20-mm [sup]2 ROI was chosen on the muscle by the spine, left lobe of the liver, head of the pancreas, and renal cortex, avoiding obvious uneven density areas and large vessel branches, to measure the CT number and its SD.
GTPs Ameliorate the Oxidative Stress of the Renal Cortex. Because of the key role of ROS in the progression of chronic kidney disease, 4-hydroxy-2-nonenal (4-HNE) and MDA were used to indicate the oxidative stress level.
Renal cortex tissues of right kidney were fixed in 10% buffered formalin, then embedded in paraffin, and sliced.
The absence of interstitial myofibroblasts in the renal cortex of female NAME rats indicates that the progressive fibrogenesis that parallels CKD progression, responsible for the gradual renal function loss, is abrogated in female animals, suggesting that the female gender presents renoprotection features.
It strongly suggests that the mechanism may be linked to the gentamicin accumulation in renal cortex through ultrafiltration, long half-life there (100 hours comparing to 30 minutes in the plasma), diffusion from the tubular lumen to the apical membranes of renal proximal tubular cells by Megalin-mediated endocytosis precipitated the renal damage.