reactive hyperaemia

reactive hyperaemia

A brief increase in blood flow following restoration of the blood supply to a part after occlusion.
Collins Dictionary of Medicine © Robert M. Youngson 2004, 2005
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Flow associated dilation (%FAD) was calculated as change in lumen diameter (LD) of brachial artery (BA) after reactive hyperaemia from reading at rest.
[7] Diameters of the femoral and brachial arteries are measured during reactive hyperaemia and after sublingual administration of glyceryl trinitrate, both procedures inducing vasodilation; former mediated by endothelium dependent mechanism whereas the latter acts directly on smooth muscle cells (Endothelium-independent).
Human cutaneous reactive hyperaemia: Role of BKCa channels and sensory nerves.
Endothelium-dependent vasodilatation was semiautomatically calculated as a percent change in arterial diameter at the baseline and during postocclusive reactive hyperaemia.
Endothelium-dependent postocclusive reactive hyperaemia in the tested arm was induced by 5-minute ischemic test as described above.
Increased metabolic demand in a region of the brain generates a rise in blood-flow, known as reactive hyperaemia. This may be due to high consumption of oxygen and glucose in the area, the generation of metabolic waste products, or the release of the neurotransmitter glutamate from active neurons.13 If the whole brain becomes hypoxic, increased build-up of metabolic waste products can cause widespread reactive hyperaemia and increased CBF, leading to a rise in ICP and secondary damage.
(8) Bone scan shows decreased uptake of the isotope over the anterosuperior region of the femoral head and is surrounded by an area of increased uptake due to reactive hyperaemia, forming a cold in hotspots, this is pathognomonic to AVN and is never seen in TOH.
The influence of local skin heating and reactive hyperaemia on skin blood flow abnormalities in patients with reflex sympathetic dystrophy (RSD).
At that time the LDF signal increased and a reactive hyperaemia (RH) response occurred in most patients.
Arterial diameters were measured at rest and during reactive hyperaemia. Reactive hyperaemia was induced by inflation of pneumatic cuff on the upper arm to 50 mm above systolic pressure followed by cuff deflation after 5 minutes.
Following this, blood flow index (BFI) was again recorded at 1st, 2nd, 3rd, 5th, 7th and 9th minute to see post occlusive reactive hyperaemia (RH).
In present study controls had baseline diameter 5.61 [+ or -] 0.54; baseline flow 661.95 [+ or -] 164.55, reactive hyperaemia flow 780.59 [+ or -] 356.57, Hyperaemia flow 168.21 [+ or -] 98.24; FMD 5.03 [+ or -] 2.80.Microalbuminurics had baseline diameter 4.39 [+ or -] 0.68; baseline flow 631.07 [+ or -] 175.56; reactive hyperaemia flow 1001.4 [+ or -] 411.84; hyperaemia flow 142.10 [+ or -] 93.59, FMD 4.64 [+ or -] 1.25.McMeuirranet al study (10), controls had baseline diameter 3.98 [+ or -] 0.16; baseline flow 117 [+ or -] 14; hyperaemia % 675 [+ or -] 73; FMD% 9.3 [+ or -] 1.8.

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