Several mechanisms have been implicated in aphakic glaucoma, including pupillary block glaucoma
, malignant glaucoma, ghost cell glaucoma, vitreous in the anterior chamber, epithelial ingrowth, and protracted inflammation; however, these are not present in a significant number of patients.
The first type is a Phacomorphic secondary angle-closure pupillary block glaucoma
due to senile cortical cataract intumescent stage.
Fibrinous uveitis is a common postoperative complication especially in children, leading to posterior synechiae, pupillary block glaucoma
, and lenticular membrane formation.
The post operative complications were posterior capsular opacification (PCO) in 153 eyes, pupillary capture of the optic of the IOL (pup cap) in 34 eyes, post operative uveitis (p o uveitis) in 26 eyes, corneal decompensation (corn decom) in three eyes, vitreous hemorrhage (vit h'age) in two eyes, secondary glaucoma (sec gl) in two eyes, decentred IOL (dec IOL) in two eyes, pupillary block glaucoma
(pup bl gl) in two eyes and retinal detachment (RD) in one eye.