Keywords: Dermoscopy, lichen planopilaris (LPP), discoid lupus erythematosus (DLE), pseudopelade of brocq (PB).
In this observational study, we included 18 patients irrespective of age and gender with clinically and histopathologically confirmed cases of lichen planopilaris (LPP), discoid lupus erythematosus (DLE), pseudopelade of Brocq (PB).
Classification of cicatricial alopecia Lymphocytic (i) Discoid lesions of lupus erythematosus (ii) Lichen planopilaris (a) Classic LPP (b) Frontal fibrosing alopecia (c) Graham Little syndrome (iii) Pseudopelade of Brocq
(iv) Central centrifugal cicatricial alopecia (v)Alopecia mucinosa (vi) Keratosis follicularis spinulosa decalvans Neutrophilic (i) Folliculitis decalvans (ii) Dissecting cellulitis Mixed cell (i) Acne keloidalis (ii) Acne necrotica (iii) Erosive pustular dermatosis Table 2: Peroxisome proliferator-activated receptors (PPARs) in human skin.
Results In our study, major causes of cicatricial alopecia were lichen planopilaris (27.5%), discoid lupus erythematosus (25%), pseudopelade of Brocq (20%), systemic lupus erythematosus (5%) followed by scleroderma, dermatomyositis, keratosis follicularis spinulosa decalvans, aplasia cutis, kerion, follicular mucinosis, pemphigus, dissecting cellulitis of scalp/ pyogenic folliculitis and acne keloidalis nuchae in 2.5% cases each.
In our study, the maximum number of cases of cicatrical alopecia were due to LPP (27.5%) followed by DLE (25%) and pseudopelade of Brocq (25%).
Pseudopelade of Brocq (PB) is a permanent progressive scarring alopecia characterized by numerous alopecic patches localized only in the scalp, that tend to coalesce into larger, irregular plaques with polycyclic borders.