BRS should be considered in the differential diagnosis of ST-segment elevation in the right precordial leads
not explainable by other conditions such as ischemia, electrolyte abnormality, or structural heart disease.
Therefore, recognizing BS and taking necessary measures in young individuals with febrile illness and ST elevations in the right precordial leads
can be life-saving.
Depolarization * Epsilon wave (reproducible low-amplitude and signals between end of QRS complex to conduction onset of the T wave) in the right abnormalities precordial leads
Eventration of the left hemidiaphragm, the result of left phrenic nerve damage from the gunshot, allows upward displacement of the gut that pushes the heart far enough to the right that leads V1 to V3 lie over the left ventricle and record complexes resembling those usually recorded from the left precordial leads
In conclusion, VT and inverted T waves in precordial leads
in sinus rhythm are the classical findings of arrhythmogenic right ventricular cardiomyopathy.
A 12-lead electrocardiogram recorded 20 minutes later with the precordial leads
on the right side of the chest showed obvious ST-segment elevation in leads [V.
Resting twelve-lead electrocardiogram showed sinus rhythm with inverted T waves in the precordial leads
Given the physical findings of a right-sided point of maximum impulse, a right-sided electrocardiogram was also performed and revealed an acute injury pattern throughout the right precordial leads
Inclusion criteria were a diagnosis of chest pain of at least 30 minutes duration and electrocardiographic ST-segment elevation of 2 mV or more in at least two contiguous precordial leads
or 1 mV or more in two contiguous extremity leads and increase in creatinine kinase-MB (CKMB) levels three times or more.
13 seconds), with broad S waves in leads I and V6, broad R waves in lead aVR, and a multiphasic (rSrs) configuration of relatively low voltage in the anterior precordial leads
The electrocardiogram shows sinus rhythm, delayed precordial R-wave progression, inverted T waves in lead I, and low to flat T waves in all of the precordial leads
Figure 1 shows that T waves in the fixed-AP model had 20 to 40% larger amplitudes in the precordial leads
V1-V3, became biphasic in lead III, and notched in leads II and aVF.