chest leads

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Related to precordial leads: ECG

chest leads

those in which the exploring electrode is on the chest overlying the heart or its vicinity.

chest leads

(chest lēdz)
A unipolar electrocardiographic lead the exploring electrode of which is on the chest overlying the heart or its vicinity; there are six standard chest leads, designated as V1-V6.
Synonym(s): precordial leads.
References in periodicals archive ?
BRS should be considered in the differential diagnosis of ST-segment elevation in the right precordial leads not explainable by other conditions such as ischemia, electrolyte abnormality, or structural heart disease.
Therefore, recognizing BS and taking necessary measures in young individuals with febrile illness and ST elevations in the right precordial leads can be life-saving.
Depolarization * Epsilon wave (reproducible low-amplitude and signals between end of QRS complex to conduction onset of the T wave) in the right abnormalities precordial leads ([V.
Eventration of the left hemidiaphragm, the result of left phrenic nerve damage from the gunshot, allows upward displacement of the gut that pushes the heart far enough to the right that leads V1 to V3 lie over the left ventricle and record complexes resembling those usually recorded from the left precordial leads.
In conclusion, VT and inverted T waves in precordial leads in sinus rhythm are the classical findings of arrhythmogenic right ventricular cardiomyopathy.
A 12-lead electrocardiogram recorded 20 minutes later with the precordial leads on the right side of the chest showed obvious ST-segment elevation in leads [V.
Given the physical findings of a right-sided point of maximum impulse, a right-sided electrocardiogram was also performed and revealed an acute injury pattern throughout the right precordial leads (Figure 2).
Inclusion criteria were a diagnosis of chest pain of at least 30 minutes duration and electrocardiographic ST-segment elevation of 2 mV or more in at least two contiguous precordial leads or 1 mV or more in two contiguous extremity leads and increase in creatinine kinase-MB (CKMB) levels three times or more.
13 seconds), with broad S waves in leads I and V6, broad R waves in lead aVR, and a multiphasic (rSrs) configuration of relatively low voltage in the anterior precordial leads.
The electrocardiogram shows sinus rhythm, delayed precordial R-wave progression, inverted T waves in lead I, and low to flat T waves in all of the precordial leads.
Figure 1 shows that T waves in the fixed-AP model had 20 to 40% larger amplitudes in the precordial leads V1-V3, became biphasic in lead III, and notched in leads II and aVF.