Chorangiosis does not occur in the postuterine
pattern of placental injury in which villous hypovascularity is typically present.
Various intensity of axial and peripheral skeleton bones growth during both uterine and postuterine
periods affects body proportions of the growing youngsters.
(PU) (postplacental) pattern of chronic hypoxic placental injury is due to primary villous changes resulting in decreased intake of oxygen from the intervillous space, as in retained stillbirth (Figure 2, C), subsets of FGR (Figure 2, D) and preeclampsia, and fetal thrombotic vasculopathy (only focally) (13,38) (Figure 2, E).
hypoxia features a homogenous placental maturity, increased smudgy syncytial knotting, increased extracellular matrix of the chorionic villi, and decreased villous cytotrophoblastic cells, Hofbauer cells, and villous vascularity (Figure 2).