Primary intervention for posthemorrhagic
hydrocephalus in very low birthweight infants by ventriculostomy.
In perinatal medicine, measurements of S100B protein in CSF have been used to monitor infants affected by perinatal asphyxia and posthemorrhagic
ventricular dilatation brain damage during cardiac surgery (60-62) (Table 1).
Volpe predicts mortality rates and incidence of progressive posthemorrhagic
hydrocephalus based on severity of the hemorrhage (Table 1).
Serial lumbar punctures for at least temporary amelioration of neonatal posthemorrhagic
The causes of death were diffuse purulent peritonitis due to injury of rectum (which was not determined during the surgical operations), fistula of pancreas, posthemorrhagic
Intraoperative evaluation with a 0 [degrees] laryngeal telescope confirmed the nature of the right posthemorrhagic
polyp (figure 2).
This discrepancy could be explained by the differences in monitoring procedures (longitudinal urine sampling from birth to 72 h, corresponding to ultrasound diagnosis of IVH in our study vs one CSF sample performed in infants when posthemorrhagic
ventricular dilatation was clearly detectable by ultrasound).
Infants with grade I hemorrhages rarely develop posthemorrhagic
hydrocephalus, approximately 25% of infants with grade II bleeds develop progressive hydrocephalus, while up to 50% of infants with grades III and IV IVH develop progressive hydrocephalus.
At the midpoint of the vibratory margin of the left vocal fold, there was a broad-based, ill-defined, fibrotic, posthemorrhagic
, vascularized mass (figure 1).