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Related to pollinosis: hay fever


an allergic reaction to pollen; hay fever.


Hay fever excited by the pollen of various plants.
Synonym(s): pollenosis
[L. pollen, pollen, + G. -osis, condition]


/pol·li·no·sis/ (pol″ĭ-no´sis) an allergic reaction to pollen; hay fever.






See hay fever.

allergic rhinitis

An inflammatory response in the nasal passages to allergens, which is the most common form of atopic-allergic disease, affecting 5–20% of the general population. Allergic rhinitis is initiated by exposure of the nasal mucosa to airborne antigens, evoking IgE production; upon repeated re-exposure to the allergen (e.g., ragweed pollen), histamine, leukotrienes C4, D4, E4, B4, PGD2, kinins, kininogen and serotonin are released.

Allergic rhinitis is the most widely used of a plethora of terms referring to the effect of allergens on the upper respiratory tract, in particular the nasopharynx. It is often related to environmental antigens—most commonly pollen—thus being known as seasonal allergic rhinitis (colloquially known as hay fever), and less often to “constant” allergens, in which case it is designated perennial allergic rhinitis.

Clinical findings
Paroxysms of sneezing, nasal congestion, nasal and ocular pruritus, tearing, rhinorrhoea, anosmia, ageusia, postnasal drip (which may cause coughing), partial or total obstruction of airflow, throat clearing, and allergic periorbital hematomas (black eyes).
Skin testing with appropriate inhalant allergens is of greater use than measuring serum IgE.
Avoid allergens; antihistamines (especially H1-receptor antagonists); sympathomimetic amines; anticholinergics; corticosteroids; decongestants; cromolyn sodium; immunotherapy.

Unclear; possibly a hypersensitivity response to allergens in pollen, dander, mites, insects, mould spores, foods; most patients have circulating IgE antibodies that bind to high-affinity receptors on mast cells and basophils, and to low-affinity receptors on other cells, evoking release of inflammatory mediators.


, pollenosis (pol'i-nō'sis, lĕ-nōsis)
Hay fever excited by the pollen of various plants.
[L. pollen, pollen, + G. -osis, condition]


an allergic reaction to pollen; hay fever.

Patient discussion about pollinosis

Q. I have chronic hayfever problems in the mornings for the first hour.Seems to be a correlation with dairy produ I also got asthma 8 years ago at age 69, after having 2 pet cats. It is controlled with 2 puffs of Symbicord daily, am & pm. Anyone managed a complete cure?

A. Hey lixuri,you mean to tell me after after 25yrs as a therapist,All my patients had to do is drink water all day.i love it,how long does it take to work,an what does the patient do in the mean time if they have a asthmatic attack(drink WAter while you cant breath?-PLEASE SEND ME AN AANSWER.---mrfoot56.

Q. Regarding Seasonal Nasal allergy. My father is suffering from seasonal nasal allergies. He took a 24-hour loratadine pill, 5 hours ago. His nose is still running just like it was. Can I take a benedryl, or is it dangerous to mix loratadine and benedryl? What else can I do to stop my nose?

A. except well known drug interactions- most Dr. check it out with a computer program they have. you need to ask a Dr. or a pharmacist about it. but i can tell you that if you wait 4 times the T1/2 - that is enough to consider the drug out of the system.

Q. is seasonal allergies are treatable?

A. here is the link to the Merck manual about that-

More discussions about pollinosis
References in periodicals archive ?
Consequently, the first article on Japanese cedar pollinosis in an international Journal was published in 1987 by Ishizaki et al.
Epidemiological trends of Japanese cedar pollinosis
When the patients with Japanese cedar pollinosis began to increase?
2) New pollinosis patients and the dynamics of airborne pollen.
Based on the two investigations described above on the year of onset of Japanese cedar pollinosis, a rapid increase in the incidence rate appears to be directly correalated with the sudden increases of Japanese cedar pollen dispersal in the 1970s (cf.
Creating these uniform plantation forests has led to a "baby-boom" generation of Japanese cedars and has caused the explosion of Japanese cedar pollinosis since the 1970s.
19) carried out a seroepidemiological study to investigate whether the prevalence of Japanese cedar pollinosis had increased during the 1970s to 1980s.
The results of the surveys, in which otolaryngologists and their family members completed questionnaires, showed that the prevalence rate of Japanese cedar pollinosis increased from 16.
The Tokyo Metropolitan Government was the first local government to establish an action committee for pollinosis in 1983 and this committee still exists.
5, indicated that the estimated prevalence rate of Japanese cedar pollinosis in Tokyo was 10.
Figure 6 shows the estimated prevalence rate of the pollinosis in different age groups of the residents involved in the epidemiological studies.
22) Since the life style and the microbial environment in Japan suddenly changed after the end of the war, the big difference in the prevalence rate of Japanese cedar pollinosis between certain age groups described above may well be due to the changes in the microbial exposure of children, together with the westernized life style after war.