polioencephalomalacia


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polioencephalomalacia

(pō'lē-ō-en-sef'ă-lō-mă-lā'shē-ă),
Cerebrocortical necrosis in sheep and goats, also seen in cattle, deer, and antelope. Associated with thiamine deficiency, water deprivation-sodium ion toxicosis, lead poisoning, and particularly with high dietary sulfur levels leading to high hydrogen sulfide and acetic acid levels in the rumen, which adversely affect carbohydrate metabolism.
References in periodicals archive ?
The intramenineal hemorrhages (not related to euthanasia) was associated with focally extensive polioencephalomalacia. Onhistopathology, a single area of the meninges showed a massive blood clot, while the subjacent gray matter was markedly rarefied.
Like most cases of polioencephalomalacia, the pathology observed in case D was most likely due to a thiamine deficiency.
Polioencephalomalacia and enterotoxemia are sub-acute results of grain overload.
Polioencephalomalacia (PEM) is a disorder of sheep and cattle that appears to be caused by the ingestion of substances that create a thiamin deficiency.
Ruminal acidosis combined with excess sulfur intake may lead to a disorder called polioencephalomalacia, which is discussed in Chapter 9.
Polioencephalomalacia (PEM) is a disorder occurring most commonly in sheep and cattle that is caused by a thiamin deficiency.