placental insufficiency


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insufficiency

 [in″sŭ-fish´en-se]
inability to perform properly an allotted function; called also incompetence.
adrenal insufficiency abnormally diminished activity of the adrenal gland; called also hypoadrenalism.
adrenocortical insufficiency abnormally diminished secretion of corticosteroids by the adrenal cortex; see also addison's disease. Called also hypoadrenocorticism and hypocorticism.
aortic insufficiency inadequate closure of the aortic valve, permitting aortic regurgitation.
coronary insufficiency decreased supply of blood to the myocardium resulting from constriction or obstruction of the coronary arteries, but not accompanied by necrosis of the myocardial cells. Called also myocardial ischemia.
ileocecal insufficiency inability of the ileocecal valve to prevent backflow of contents from the cecum into the ileum.
mitral insufficiency inadequate closure of the mitral valve, permitting mitral regurgitation.
placental insufficiency dysfunction of the placenta, with reduction in the area of exchange of nutrients; it often leads to fetal growth retardation.
pulmonary valve insufficiency inadequate closure of the pulmonary valve, permitting pulmonic regurgitation.
respiratory insufficiency see respiratory insufficiency.
thyroid insufficiency hypothyroidism.
tricuspid insufficiency incomplete closure of the tricuspid valve, resulting in tricuspid regurgitation.
valvular insufficiency failure of a cardiac valve to close perfectly, causing valvular regurgitation; see also aortic, mitral, pulmonary, and tricuspid insufficiency.
velopharyngeal insufficiency inadequate velopharyngeal closure, due to a condition such as cleft palate or muscular dysfunction, resulting in defective speech.
venous insufficiency inadequacy of the venous valves and impairment of venous return from the lower limbs (venous stasis), often with edema and sometimes with stasis ulcers at the ankle.
References in periodicals archive ?
This finding was to be expected, as raised RIs are associated with placental insufficiency and growth restriction.
IUGR with placental insufficiency was defined by fetal weight below the 10th percentile for gestational age in combination with abnormal Doppler indices (either umbilical artery RI > 95th centile, middle cerebral artery RI < 5th centile, or uterine artery RI > 95th centile for gestational age).
This condition termed placental insufficiency leads to slowed or even arrested fetal umbilical artery diastolic flow compromising maternal/fetal exchange, causing fetal hypoxemia and acidosis.
Recurrent miscarriage and other placental insufficiency occur when the placenta does not develop properly, or is damaged.
The fetal origins hypothesis: placental insufficiency and inheritance versus maternal malnutrition in well-nourished populations.
The revealed morphological changes underlie the development of chronic placental insufficiency in 74%.
Reduced protein synthesis caused by ER stress has a severe detrimental effect on placental development by causing decreased levels of many hormones, growth factors, and regulatory proteins--leading to the placental insufficiency and dysfunction of preeclampsia.
Placental insufficiency due to developmental pathologic changes in the placental vasculature results in damaged exchange of gas and nutrients as well as placental size reduction [1].
One local example of why PATH has quietly invested over R3.5 million in MRC activities over the past 3 years is a Stellenbosch University medical school-developed device called the 'Umbiflow', a mobile and highly versatile Doppler waveform analyser measuring placental insufficiency in small-for-gestational-age babies in the third trimester.
Arsenic exposure can lead to abortion due to aberrant placental vasculogenesis and placental insufficiency (Wenjie et al., 2007).In the present study PCCs were first isolatedand later effect of arsenic, cadmium and lead was observed on proliferation and morphology of cells, with an objective to assess the damage done to thesecells during gestation, which may end up with some teratological abnormalities.
To qualify, a woman must have had spontaneous abortions at less than 10 weeks' gestation in at least three or more consecutive pregnancies, or a second- or third-trimester pregnancy loss of a normal fetus, or premature birth of a morphologically normal fetus before week 34 due to preeclampsia or placental insufficiency.
The ObGyn was negligent in failing to diagnose fetal growth restriction caused by placental insufficiency. The fetal monitor showed an abnormal heart rate during that 3-hour span.