peroxisomes


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Related to peroxisomes: Glyoxysomes

peroxisomes

Membrane-bound cell organelles containing oxidative enzymes concerned in the detoxification of various molecules and in the breakdown of fatty acids to acetyl-CoA.
References in periodicals archive ?
The expression levels of 10 genes: retinoid X receptor alpha (RXRA), peroxisome proliferator-activated receptor gamma (PPARG), phospholipid transfer protein (PLTP), stearoylCoA desaturase (SCD), nuclear receptor subfamily 1 group H member 3 (NR1H3), fatty acid binding protein 3 (FABP3), carnitine palmitoyltransferase II (CPT2), acyl-Coenzyme A dehydrogenase long chain (ACADL), acyl-Coenzyme A oxidase 2 branched chain (ACOX2), and fatty acid binding protein 4 (FABP4), showed significant differences in gene expression between the low- and high-marbled groups (p < 0.
BACKGROUND: Trichloroacetic acid, an oxidative metabolite of trichloroethylene (TRI), is a ligand of the peroxisome proliferator-activated receptor [alpha] (PPAR) [alpha], which is involved in lipid homeostasis and anti-inflammation.
The paper establishes a new function for peroxisomes as a cellular compartment that promotes a rapid response to viral infection.
Studying peroxisomes with an eye to differences also allowed Novikoff to discover microperoxisomes in 1972.
Direct demonstration that the deficient oxidation of very long chain fatty acids in X-linked adrenoleukodystrophy is due to an impaired ability of peroxisomes to activate very long chain fatty acids.
Understanding peroxisome biology at the molecular level and how peroxisomes communicate with the rest of the cell will help to develop both treatments and methodologies to treat disease.
2006) and PPAR[alpha]-dependent induction of hepatic peroxisome proliferation by phthalates (Lapinskas et al.
says the eviden ce suggests that the peroxisomes in human liver cells don't proliferate markedly in response to the drugs.
Induction of cytosolic and microsomal epoxide hydrolases and proliferation of peroxisomes and mitochondria in mouse liver after dietary exposure to p-chlorophenoxyacetic acid, 2,4-dichloro-phenoxyacetic acid and 2,4,5-trichlorophenoxyacetic acid.
Within minutes to hours of PPC exposure, the genetic trio stimulates a cell's peroxisome production of H.
Initially, it was hypothesized that the proliferation of peroxisomes themselves was causally related to cancer (e.
They are glycogen poor from the start with increased numbers of mitochondria, peroxisomes, and ribosomes.

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