peribronchiolar

peribronchiolar

 [per″ĭ-brong″ke-o´ler]
around the bronchioles.

per·i·bron·chi·o·lar

(per'i-brong'kē-ō'lăr),
Surrounding the bronchioles.
References in periodicals archive ?
When accompanied by peribronchiolar metaplasia, centrilobular fibrosis strongly suggests prior small airway injury, perhaps due to chronic HP, and a careful search for residual granulomas or giant cells should be undertaken.
As shown in Figure 4(b), asthma group mice showed prominent neutrophil infiltration of the peribronchiolar and perivascular connective tissues, together with the hypersecretion of mucus by epithelial cells.
We observed peribronchiolar inflammation 1 day after Cr(VI) exposure, which increased up to 8 days after intranasal treatment (Figure 1D).
Histologically, the OP pattern is characterized by a temporally uniform proliferation of intra-alveolar organizing fibroblastic tissue in a patchy peribronchiolar distribution.
Not much is known about the role of macrophages in this part of the disease, but pigmented macrophages were found to cluster around small airways and these were associated with peribronchiolar fibrosis [81].
Mexico City dogs exhibit focal peribronchiolar inflammatory infiltrates that surround the adjacent blood vessels, some of which contain platelet thrombi and marginated neutrophils (CalderOin-Garciduenas et al.
Scores for peribronchiolar inflammatory cell infiltrates in sections from each lung were graded; a score of 0 indicated the absence of inflammatory cell infiltrates; a score of 1, less than five layers of inflammatory cells in <50% of the bronchiolar submucosa; 2, more than five layers of inflammatory cells in <50% of the bronchiolar submucosa; 3, less than five layers of inflammatory cells in >50% of the bronchiolar submucosa; and 4, more than five layers of inflammatory cells in >50% of the bronchiolar submucosa [18].
This technique provides for a wide dissemination of particles in a peribronchiolar pattern within the alveolar region (Rao et al.
Detailed morphometric analysis has extensively expanded our knowledge of airway alterations in both membranous and respiratory bronchioles, (14) and it has documented airway narrowing with inflammation and fibrosis, and loss of the peribronchiolar alveolar attachments.
At 14 days after infection, we observed diffuse granulomatous pneumonia and the severe destruction of lung parenchyma with the peribronchiolar and perivascular infiltration of macrophages and neutrophils (Figure 5(h)).