sup] At present, there is no effective rehabilitation therapy for ocular motor
nerve palsy caused by brain surgery or trauma.
Joubert syndrome" revisited: Key ocular motor
signs with magnetic resonance imaging correlation.
Due to the high incidence of ocular motor
dysfunction, which can affect ocular fixation, pursuit, and saccades, careful assessment of these functions should be undertaken (see Figures 3 and 4).
nerve palsies: Dr Sandeep Chattopadhyay
External ocular motor
palsies involving third, fourth and sixth cranial nerves are frequent in acute HZO if carefully evaluated but are usually self limiting.
This is, due to the important role played by the cerebellum in visual and visual-spatial processing; different cerebellar regions are implicated in ocular motor
function and the associated involvement of the ocular structures in the cerebellar diseases.
Common symptoms of multiple sclerosis include visual problems, ocular motor
abnormality, paresthesia, muscle weakness, spasticity, urinary dysfunction, and cognitive impairment.
Among the topics are color vision and color blindness, slit-lamp examination, corneal confocal microscopy, optic disc assessment in glaucoma, indocyanine green angiography, A-scan and B-scan ultrasonography, electrophysical tests for assessing visual function, anterior segment optical coherence tomography, localizing an intra-ocular foreign body, diagnostic procedures for genetically transmitted eye diseases, diagnostic modalities in intra-ocular malignancies, and ocular motor
And outside the vestibular system, ocular motor
impairments--such as impairments in eye tracking and movement--are also common, some experts say.
It is a motivating way to exercise and improve ocular motor
skills of visual tracking and scanning, visual endurance, and eye-hand coordination.
Among the classic clinical findings of JS are hypotonia, ataxia, mental-motor retardation, and respiratory findings such as apnoea/hyperpopnoea, and opthalmological findings as ocular motor
Imaging of ocular motor
nerves in the brainstem revealed bilateral absence of CN6 and bilateral relatively hypoplastic CN7 along its course at the ponto-medullary junction (Figure 3).