noradrenergic


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Related to noradrenergic: Noradrenergic system

noradrenergic

 [nor″ah-dren-er´jik]
activated by or secreting norepinephrine.

noradrenergic

(nôr′ăd-rə-nûr′jĭk)
adj.
Stimulated by or releasing norepinephrine: noradrenergic neurons.

nor′ad·re·ner′gi·cal·ly adv.
References in periodicals archive ?
Waterhouse, "Evidence for broad versus segregated projections from cholinergic and noradrenergic nuclei to functionally and anatomically discrete subregions of prefrontal cortex," Frontiers in Behavioral Neuroscience, vol.
This acupuncture treatment decreased the frequency of the wave from small-amplitude faster waves to large-amplitude slow waves, which demonstrate that acupuncture stimulation may change the state from light anesthesia to deep anesthesia, and also decreased the activity of noradrenergic neurons in the LC.
(29) demonstrated that amplifying the noradrenergic response to stress by means of yohimbine treatment prior to submission to acute stress released galanin in central nucleus of the amygdala, which produced an anxiolytic response in EPM test.
Hence, in the present study, scopoletin and rutin were used to establish its interaction with dopaminergic and noradrenergic systems.
Central antinociceptive effects of mitragynine in mice contribution of descending noradrenergic and serotonergic systems.
The TCAs amitriptyline (with both serotonergic and noradrenergic effects) and desipramine (with predominantly noradrenergic effects) have shown efficacy in the treatment of diabetic neuropathy.[11] Clomipramine, a TCA with potent serotonergic properties, has shown clinical response in the treatment of diabetic neuropathy.[12] The selective serotonin reuptake inhibitors (SSRIs) have also been shown to elicit pain relief in patients with diabetic neuropathy in some,[13,14] but not all[11] studies.
Active behaviors in the rat forced swimming test differentially produced by serotonergic and noradrenergic antidepressants.
While 5-HT in the descending modulating system can inhibit pain transmission ascending to the brain from the periphery, it appears that an intact noradrenergic system is necessary for the inhibitory influences of the serotonergic system to be appreciated.
"In theory we should have some really simple ways to prevent PTSD from occurring if we get in there soon enough: reducing noradrenergic overactivity via [alpha.sub.2]-adrenergic receptor agonism with an agent such as Clonidine; postsynaptic beta-adrenergic blocking with a drug such as propranolol; or [alpha.sub.1]-adrenergic receptor blocking, as with prazosin.
"In theory we should have some really simple ways to prevent PTSD from occurring if we get in there soon enough: reducing noradrenergic overactivity via alph[a.sub.2]-adrenergic receptor agonism with an agent such as Clonidine; postsynaptic beta-adrenergic blocking with a drug such as propranolol; or alph[a.sub.1]-adrenergic receptor blocking, as with prazosin.
In fact, depression and anxiety may increase the perception of acute and chronic pain.[7,8] The serotonergic and noradrenergic neuronal pathways have been clearly implicated in the etiology of depression as well as in pain modulation in the central nervous system.
Attention and emotion have a positive impact on memory formation, which is related to the activation of the noradrenergic system in the brain (1,2).