neuronal plasticity

neuronal plasticity

Neurophysiology
1. The ability of neurons to stabilize or alter synapses.
2. The malleability of cortical representations of sensory and motor innervation, which has a range of 10-14 mm in the somatosensory cortex in animal models that have undergone long-term loss of sensory nerves in the forelimbs. See Long-term potentiation, Synaptic plasticity.
References in periodicals archive ?
It plays an important role in various aspects of neuronal plasticity, such as neurogenesis, learning and memory, long-term potentiation (LTP), and mood changes.
7 Given that the most noted expression of BDNF and its robust correlated receptor, TrkB (Thyrozine Kinase Type-B), was found in brain tissues, such as hippocampus and cerebellum indicating that BDNF may modify neuronal plasticity in adult animals;9 more importantly BDNF operates upon the reward system of the brain, which has a main role in drug dependency;10 thus BDNF is considered to be a better candidate for mediating the long-term benefits of exercise on the brain7 and a candidate locus correlation with drug dependence.
Sleep enables neuronal plasticity processes, which are required for stroke recovery.
Objective: Neuronal plasticity is an important mechanism for memory and cognition, and also fundamental to fine-tune perception to the environment.
Thus, hyperoxia-triggered changes in neuronal plasticity gene expression were significantly ameliorated by single Epo treatment.
The organization of receptor placement creates different wave properties that are relevant to the formation of neuronal plasticity.
Antidepressantlike effects of 3,6'-disinapoyl sucrose on hippocampal neuronal plasticity and neurotrophic signal pathway in chronically mild stressed rats.
Everitt, joint winner of the prestigious Fondation (CORR) Ipsen Neuronal Plasticity Prize for 2014, explained his research which involves targeting "memory plasticity.
According to the study, these distinct adaptations are because of the neuronal plasticity of the brain, which allows us to adapt to environmental stimuli.
Nutritional deficiencies have been shown to influence neuronal function and produce defects in neuronal plasticity, as well as impact behavior in children with attention deficit hyperactivity disorder (ADHD).
Catenin/aN-Catenin are molecular targets in the neuroprotection and neuronal plasticity mediated by atorvastatin after focal cerebral ischemia.

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