neurohumoral transmission

(redirected from neurohumoral)
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neu·ro·hu·mor·al trans·mis·sion

a process by which a presynaptic cell, on excitation, releases a specific chemical agent (a neurotransmitter) to cross a synapse to stimulate or inhibit the postsynaptic cell.
Synonym(s): neurotransmission
References in periodicals archive ?
Neurohumoral mechanisms of acute exercise-induced gastric emptying delay.
The goal of pharmacologic treatment at this stage is to inhibit angiotensin, aldosterone, and catecholamines, and increase salt excretion to prevent long-term damage resulting from the body's sustained neurohumoral and inflammatory compensatory mechanisms (Huether & McCance, 2017).
[30] Based on the differences in the effects on the neurohumoral factors between cilnidipine and other drugs, it can be speculate that the inhibitory effect of cilnidipine on the renin-angiotensin-aldosterone system by its N-type calcium channel blocking action [31-34] may have decreased the suppression of the K+ channels.
The influence of physical activity on ABI among activity individuals is linked to the physiologic adjustments and adaptations that improve endothelial and neurohumoral functioning (40).
Furthermore the endothelium responds to the numerous intrinsic factors including temperature, sheer stress and transmural pressure as well as extrinsic factors such as mental stress, medication and neurohumoral responses.
The natriuretic peptides (NPs) are markers of hemodynamic stress on the heart, denoting the neurohumoral activation of the myocardium.
La fisiopatologia se relaciona con hipoperfusion renal que genera un estimulo neurohumoral con activacion del eje renina-angiotensina-aldosterona.
Lima et al., "Neurohumoral features of myocardial stunning due to sudden emotional stress," New England Journal of Medicine, vol.
Additionally, improved functional capacity of the patients and reduced neurohumoral activation were observed [17].
Progression of heart failure leads to chronic activation of neurohumoral regulation which promotes increase in atrial natriuretic peptide, brain natriuretic peptide, angiotensin II, and aldosterone all which contribute to left atrial remodeling [12].
[1] In addition to structural renal abnormalities related to hypertension, diabetes or atherosclerosis, renal dysfunction in HF patients may result from renal hypoperfusion caused by haemodynamic, neurohumoral and inflammatory factors.
The role of genetic variants in genes regulating the oxytocin-vasopressin neurohumoral system in childhood-onset aggression.