myeloid


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myeloid

 [mi´ĕ-loid]
1. pertaining to, derived from, or resembling bone marrow.
2. pertaining to the spinal cord. defs. 1 and 2 called also medullary.
3. having the appearance of myelocytes, but not derived from bone marrow.
Miller-Keane Encyclopedia and Dictionary of Medicine, Nursing, and Allied Health, Seventh Edition. © 2003 by Saunders, an imprint of Elsevier, Inc. All rights reserved.

my·e·loid

(mī'ĕ-loyd),
1. Pertaining to, derived from, or manifesting certain features of the bone marrow.
2. Sometimes used with reference to the spinal cord.
3. Pertaining to certain characteristics of myelocytic forms, but not necessarily implying origin in the bone marrow.
[myel- + -oid]
Farlex Partner Medical Dictionary © Farlex 2012

myeloid

(mī′ə-loid′)
adj.
1. Of, relating to, or derived from the bone marrow.
2. Of or relating to certain blood cells, including granulocytes, monocytes, and sometimes erythrocytes and platelets but excluding lymphocytes, that are thought to derive from a common progenitor in the bone marrow.
3. Of or relating to the spinal cord.
The American Heritage® Medical Dictionary Copyright © 2007, 2004 by Houghton Mifflin Company. Published by Houghton Mifflin Company. All rights reserved.

myeloid

adjective 
1. Referring to the BM.
2. Referring to granulocytes and maturation thereof, aka myelogenous.
3. Referring to the spinal cord–rarely used in practice.
McGraw-Hill Concise Dictionary of Modern Medicine. © 2002 by The McGraw-Hill Companies, Inc.

my·e·loid

(mī'ĕ-loyd)
1. Pertaining to, derived from, or manifesting certain features of the bone marrow.
2. Sometimes used with reference to the spinal cord.
3. Pertaining to certain characteristics of myelocytic forms, but not necessarily implying origin in the bone marrow.
[myel- + -oid]
Medical Dictionary for the Health Professions and Nursing © Farlex 2012

myeloid

Pertaining to the bone marrow.
Collins Dictionary of Medicine © Robert M. Youngson 2004, 2005

Patient discussion about myeloid

Q. Are there any specific treatments for acute myeloid leukemia caused by myelofibrosis? My husband was diagnosed with these conditions. His oncologist believes his AML was caused by his myelofibrosis. (scarring of the bone marrow) This was shown on his bone marrow biopsy. Myelofibrosis is a myeloproliferative disorder. Meaning certain blood components, such as WBCs, RBCs, platelets increase significantly in the marrow.

A. Thanks for taking the time to reply to me. I'll see about inquiring his oncologists.
Lynda

More discussions about myeloid
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References in periodicals archive ?
The involvement of the chromosome 8 in myeloid leukemias suggests that dosage sensitive, cell type specific genes are localized on chromosome 8.
They are uncommon in de novo AML (6.5%), but more frequent in AML arising from a prior myeloid neoplasm (up to 30%) (41-43).
Keeping in view its importance, molecular analysis for NPM1 and FLT3-ITD mutations should be included in the initial workup of all acute myeloid leukaemia patients.
In this study, investigators looked at the integrin CD11b, which I present on myeloid cells and normally helps myeloid cell migration and its ability to fight disease.
Acute myeloid leukemia patients with CEBPA mutations have a favorable clinical outcome, but recent data indicate the favorable outcome is limited to those with double mutations.
The presence of the BCR-ABL1 fusion gene in CML is an essential feature not only for establishing diagnosis but also for monitoring response to treatment.7 Classically, in majority of the patients with chronic myeloid leukaemia, this BCR-ABL1 fusion gene results from a reciprocal translocation involving the long arms of chromosome 9 and chromosome 22.
IDH1 and IDH2 Mutation: IDH is an essential enzyme in cell metabolism, and gain of function mutations in IDH leads to DNA methylation and impaired myeloid differentiation (26).
Nodal and extranodal tumor-forming accumulation of plasmacytoid monocytes/interferon-producing cells associated with myeloid disorders.
Herein, we employed clodronate liposome (Clod Lipo) and anti-Gr-1 mAb strategies to modulate the presence and function of [Gr-1.sup.+] bone marrow-derived myeloid populations in bleomycin-challenged mice.
Immature myeloid cells or extramedullary proliferation of myeloblasts are present in MS [2].