motor endplate


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mo·tor end·plate

the large and complex end formation by which the axon of a motor neuron establishes synaptic contact with a skeletal muscle fiber (cell); several terminal branches of a motor axon end in irregular, club-shaped synaptic end formations, each of which is bedded in its own troughlike depression of the muscle fiber's surface; the postsynaptic membrane, the sarcolemma that forms the bottom of the trough, is greatly increased in surface area by deep infoldings protruding into the underlying cytoplasm of the muscle fiber; the subsynaptic interval between the plasma membrane of the axon terminals and the sarcolemma is filled with an amorphous substance; the trough is closed off toward the surface by the Schwann sheath, which peels away from the axons as the latter enter the trough and thus forms a lid over the trough; the slight bulge of this closure plate corresponds to Doyère eminence.
Synonym(s): sole-plate ending

mo·tor end·plate

(mō'tŏr end'plāt)
The large and complex end-formation by which the axon of a motor neuron establishes synaptic contact with a striated muscle fiber (cell).

motor endplate

The point of junction of a motor nerve fibre and a muscle fibre. The motor endplate is a modified area of the muscle fibre membrane at which a synapse occurs. A motor nerve axon ending may have up to 50 synaptic knobs (boutons) but a single muscle fibre has only one endplate. The neurotransmitter is acetylcholine.

motor endplate

see ENDPLATE, MOTOR.

mo·tor end·plate

(mō'tŏr end'plāt)
The large and complex end-formation by which the axon of a motor neuron establishes synaptic contact with a striated muscle fiber (cell).
References in periodicals archive ?
Simons, "Do endplate noise and spikes arise from normal motor endplates?" The American Journal of Physical Medicine and Rehabilitation, vol.
(9) The motor endplates with which the nerve communicates will eventually cease to function in 12-18 months.
Simons, "Do endplate noise and spikes arise from normal motor endplates?" American Journal of Physical Medicine & Rehabilitation, vol.
During this period, we can note the disappearance of motor endplates and as a consequence, muscle atrophy, and after that, fibrosis.
Rehabilitation of the facial nerve and subsequent reinnervation of the mimetic motor endplates are achieved through axonal growth.
However, Greene proposed a standard hypothesis about dysfunctional motor endplates in 2001.
A modified Karnovsky and Roots method was used to detect the activity of AChE in the NMJ.[sup][12] A muscle strip containing motor endplates was removed from the ventral costal region of the left hemidiaphragm and blotted dry.