Chiang, "Activation of
lytic cycle of Epstein-Barr virus by suberoylanilide hydroxamic acid leads to apoptosis and tumor growth suppression of nasopharyngeal carcinoma," International Journal of Cancer, vol.
We believe that during the strongest rainfall levels (91-195 mm [month.sup.-1]) since September to November 2004 and April 2005, the entrance of allochthonous substrates by run-off or river discharges might stimulate the induction of
lytic cycle in lysogenic prokaryotes or favor the exportation of certain allochthonous populations from land (e.g., Bergstrom & Jansson, 2000; Hewson et al., 2012; Williamson et al., 2014).
Sinclair, "The Epstein-Barr virus
lytic cycle activator Zta interacts with methylated ZRE in the promoter of host target gene egr1," Journal of General Virology, vol.
Furthermore, proteins encoded by lysis-cassette genes (as holins and endolysins), which are responsible for progeny release during the phage
lytic cycle, are arousing growing interest too.
The prophage
lytic cycle can be induced with DNA damaging agents (23); therefore, to address whether the toxin carried in [stx.sub.1a]-encoding S.
Ironically, NF-[kappa]B activation is associated with viral latency, whereas its inhibition results in viral entry into the
lytic cycle [17, 18].
1) at basal conditions, after
lytic cycle enhancement, and after treatment by the [alpha]-amanitin RNA polymerase transcription inhibitor (16-18).
Recent studies from our group have shown that coculture of PEL B-cell lines (which are latently infected with HHV-8) with CD4+ T cells infected with HIV-1 results in induction of HHV-8
lytic cycle replication in the B-cell line?
Ooka, "The
lytic cycle of Epstein-Barr virus in the nonproducer Raji line can be rescued by the expression of a 135-kilodalton protein encoded by the BALF2 open reading frame," Journal of Virology, vol.
The viral
lytic cycle was induced by incubating BCBL-1 cells with 20 ng/mL of 12-Otetradecanoylphorbol-13-acetate (TPA) (Sigma, St.
For example, the lysogenic state of phage lambda is maintained by the cI repressor gene, which binds to control regions for the
lytic cycle, shutting down those regions.
We have previously demonstrated a limited or defective T-cell response against EBV antigens in SLE patients, showing a reduced number of T-cells becoming activated and producing IFN[gamma] upon EBV stimulation (both the latent antigen EBNA1 and the
lytic cycle antigen EBV early antigen diffuse (EBV-EA/D)) [25].