Chiang, "Activation of lytic cycle
of Epstein-Barr virus by suberoylanilide hydroxamic acid leads to apoptosis and tumor growth suppression of nasopharyngeal carcinoma," International Journal of Cancer, vol.
We believe that during the strongest rainfall levels (91-195 mm [month.sup.-1]) since September to November 2004 and April 2005, the entrance of allochthonous substrates by run-off or river discharges might stimulate the induction of lytic cycle
in lysogenic prokaryotes or favor the exportation of certain allochthonous populations from land (e.g., Bergstrom & Jansson, 2000; Hewson et al., 2012; Williamson et al., 2014).
Sinclair, "The Epstein-Barr virus lytic cycle
activator Zta interacts with methylated ZRE in the promoter of host target gene egr1," Journal of General Virology, vol.
Furthermore, proteins encoded by lysis-cassette genes (as holins and endolysins), which are responsible for progeny release during the phage lytic cycle
, are arousing growing interest too.
The prophage lytic cycle
can be induced with DNA damaging agents (23); therefore, to address whether the toxin carried in [stx.sub.1a]-encoding S.
Ironically, NF-[kappa]B activation is associated with viral latency, whereas its inhibition results in viral entry into the lytic cycle
1) at basal conditions, after lytic cycle
enhancement, and after treatment by the [alpha]-amanitin RNA polymerase transcription inhibitor (16-18).
Recent studies from our group have shown that coculture of PEL B-cell lines (which are latently infected with HHV-8) with CD4+ T cells infected with HIV-1 results in induction of HHV-8 lytic cycle
replication in the B-cell line?
Ooka, "The lytic cycle
of Epstein-Barr virus in the nonproducer Raji line can be rescued by the expression of a 135-kilodalton protein encoded by the BALF2 open reading frame," Journal of Virology, vol.
The viral lytic cycle
was induced by incubating BCBL-1 cells with 20 ng/mL of 12-Otetradecanoylphorbol-13-acetate (TPA) (Sigma, St.
For example, the lysogenic state of phage lambda is maintained by the cI repressor gene, which binds to control regions for the lytic cycle
, shutting down those regions.
We have previously demonstrated a limited or defective T-cell response against EBV antigens in SLE patients, showing a reduced number of T-cells becoming activated and producing IFN[gamma] upon EBV stimulation (both the latent antigen EBNA1 and the lytic cycle
antigen EBV early antigen diffuse (EBV-EA/D)) .