lipoperoxide

lipoperoxide

Pathophysiology A peroxide–an O-O containing free radicals–which has peroxidized the carbon atoms close to the double bonds in an unsaturated fatty acid. See Peroxide.
References in periodicals archive ?
The present study aimed to evaluate the physiological behavior, the activity of antioxidant enzymes and the levels of lipoperoxide in seeds of Peltophorum dubium (Spren10 gel) Taubert when subjected to immersion in heated water in order to overcome physical dormancy.
Yagi, "A simple fluorometric assay for lipoperoxide in blood plasma," Biochemical Medicine, vol.
In a similar animal study the increased levels of IL6, IL8 and LPO (lipoperoxide) in smokers with chronic bronchitis is compared with non-smokers.
Yagi, "A simple fluorometric assay for lipoperoxide in blood plasma, " Biochemical Medicine, vol.15, no.2, pp.212-216, 1976.
A GR blockade may exacerbate the pathological and pathophysiological changes of endotoxemia: (1) faster drop in arterial blood pressure, (2) more severe pathological changes involving multiple organs, (3) increased of leukocyte adherence in venules and more pronounced rheological changes in the mesenteric microcirculation, and (4) striking elevation of serum acid phosphatase, phospholipase [A.sub.2] activity, and lipoperoxide (Fan et al.
Activation of peroxisome proliferator-activated receptor alpha by dietary fish oil attenuates steatosis, but does not prevent experimental steatohepatitis because of hepatic lipoperoxide accumulation.
Signs of altered oxidation have been detected in sera of patients with IgAN, including increased levels of lipoperoxide or malondialdehyde and reduced activity of superoxide dismutase, catalase, and glutathione peroxidase [26].
Studies on lipoperoxide of normal pregnant women and of patients with toxaemia of pregnancy.
Effects of silybin on production of oxygen free radical, lipoperoxide and leukotrienes in brain following ischemia and reperfusion.
Using necrotic models of hepatic cells in animal experiments, Rosser and Gores (1995) demonstrated the relationship between advancing hepatic dysfunction and lipoperoxide reaction (Rosser and Gores 1995).
Along with its role in Pb-induced hepatotoxicity, oxidative stress was also noted to play a significant role in the regression phase of hepatic hyperplasia with the generation of lipoperoxide (LPO) and other oxidants and the induced expression of cytokine mediators, including TNF-[alpha].