Three diabetics had lichen amyloidosis and one had macular amyloidosis.
Among the 20 clinically lichen amyloidosis patients, 85% had used scrubs.
Among the 29 scrub users, clinically, 17 (58.6%) developed lichen amyloidosis, 7 (24.1%) developed macular amyloidosis and remaining 5 (17.2%) biphasic amyloidosis whereas, among the 9 non-users, macular amyloidosis was seen most frequently (44.4%); lichen amyloidosis in 33.3% and biphasic amyloidosis, 22.2%.
When small focal deposits were seen in the papillary dermis, macular amyloidosis was diagnosed and wherever globular amyloid deposits were seen, which displaced the rete ridges, a diagnosis of lichen amyloidosis was made.
demonstrated that tanscutaneous electrical nerve stimulation (TENS) therapy was quite effective treating pruritis in patients with macular amyloidosis.13 In an another study recently reported menthol was used effectively in the treatment of therapy resistant pruritus in lichen amyloidosis.14 There are not many reports about the usage of topical calcineurin inhibitors, however Cazares et al reported improvement in lichen amyloidosis using treatment with 0.1% topical tacrolimus ointment.15
Patients with lichen amyloidosis were treated with UVB or topical PUVA on one limb and moderate to potent topical corticosteroids on the other limb.
Primary localized cutaneous amyloidosis (PLCA) (cutaneous lichen amyloidosis and macular amyloidosis).