auris was cultured from the following sites: blood (45 isolates), urine (11), respiratory tract (eight), bile fluid (four), wound (four), central venous catheter tip (two), bone (one), ear (one), and a
jejunal biopsy (one).
Intestinal antibody pattern of celiac disease: occurrence in patients with normal
jejunal biopsy histology.
Ehrmann et al (15) have studied apoptotic markers in
jejunal biopsy specimens of active and potential celiac disease and have suggested the roles of Fas and/or Fas-L, Bcl2, and tissue transglutaminase in villous atrophy.
Of these 15 patients, 13 patients had
jejunal biopsy which all showed positive histopathological changes indicative of CD.
The patients were undergone stool examination, proctosigmoidoscopy, colonic swab of Entamoeba histolytica, faecal fat excretion, D-Xylose absorption test, serum proteins and
jejunal biopsy.
Upon presentation to the surgery department, the presented case was first diagnosed as leukemic infiltration; however,
jejunal biopsy showed lymphoma.
A
jejunal biopsy identified mucosal atrophy, markedly shortened villi, and hypertrophy of the crypts.
The patient was an IgA-deficient, 47-year-old white woman with chronic diarrhea who was diagnosed with celiac disease on the basis of a
jejunal biopsy that showed subtotal villous atrophy, a strongly positive antiendomysium IgG antibody, and abnormal intestinal permeability.
The standard medical test for coeliacs is a microscopic inspection of the small intestine in a process known as a
jejunal biopsy.
All 80 patients underwent
jejunal biopsy. Celiac disease was diagnosed in 48 of 80 patients on the basis of the revised ESPGAN criteria (4); therefore, the total CD patients reached 122 cases in this study.
Silent coeliac disease: patients are asymptomatic but have villous atrophy on
jejunal biopsy.
Observer variation in assessment of
jejunal biopsy specimens.